VIAF

Virtual International Authority File

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Leader 00000nz a2200037n 45 0
001 WKP|Q56696607 (VIAF cluster) (Authority/Source Record)
003 WKP
005 20241121000040.0
008 241121nneanz||abbn n and d
035 ‎‡a (WKP)Q56696607‏
024 ‎‡a 0000-0003-3815-0891‏ ‎‡2 orcid‏
024 ‎‡a 55499938500‏ ‎‡2 scopus‏
035 ‎‡a (OCoLC)Q56696607‏
100 0 ‎‡a Jesse Williams‏ ‎‡c wetenschapper‏ ‎‡9 nl‏
400 0 ‎‡a Jesse Williams‏ ‎‡c researcher‏ ‎‡9 en‏
670 ‎‡a Author's A Polecat's View of Patrolling Monocytes.‏
670 ‎‡a Author's A Stromal Niche Defined by Expression of the Transcription Factor WT1 Mediates Programming and Homeostasis of Cavity-Resident Macrophages‏
670 ‎‡a Author's Allergen Exposure in Lymphopenic Fas-Deficient Mice Results in Persistent Eosinophilia Due to Defects in Resolution of Inflammation‏
670 ‎‡a Author's B Cell-Mediated Antigen Presentation through MHC Class II Is Dispensable for Atherosclerosis Progression‏
670 ‎‡a Author's Brown adipose tissue monocytes support tissue expansion‏
670 ‎‡a Author's CCR7 and IRF4-dependent dendritic cells regulate lymphatic collecting vessel permeability‏
670 ‎‡a Author's Cloning and expression of a zebrafish 5-HT‏
670 ‎‡a Author's Cloning and expression of a zebrafish 5-HT(2C) receptor gene.‏
670 ‎‡a Author's Contribution of metabolic disease to bone fragility in MAGP1-deficient mice.‏
670 ‎‡a Author's Cytokine Circuits in Cardiovascular Disease‏
670 ‎‡a Author's Fas ligand expression on T cells is sufficient to prevent prolonged airway inflammation in a murine model of asthma.‏
670 ‎‡a Author's Homeostatic Control of Innate Lung Inflammation by Vici Syndrome Gene Epg5 and Additional Autophagy Genes Promotes Influenza Pathogenesis‏
670 ‎‡a Author's ICOS-expressing lymphocytes promote resolution of CD8-mediated lung injury in a mouse model of lung rejection‏
670 ‎‡a Author's ICOS protects against mortality from acute lung injury through activation of IL-5+ ILC2s‏
670 ‎‡a Author's IL-33-dependent induction of allergic lung inflammation by FcγRIII signaling.‏
670 ‎‡a Author's Limited Macrophage Positional Dynamics in Progressing or Regressing Murine Atherosclerotic Plaques-Brief Report‏
670 ‎‡a Author's Limited proliferation capacity of aortic intima resident macrophages requires monocyte recruitment for atherosclerotic plaque progression‏
670 ‎‡a Author's Macrophage Biology, Classification, and Phenotype in Cardiovascular Disease: JACC Macrophage in CVD Series (Part 1)‏
670 ‎‡a Author's MHC II+ resident peritoneal and pleural macrophages rely on IRF4 for development from circulating monocytes‏
670 ‎‡a Author's Monocyte Recruitment, Specification, and Function in Atherosclerosis‏
670 ‎‡a Author's Myocardial B cells are a subset of circulating lymphocytes with delayed transit through the heart‏
670 ‎‡a Author's Neutrophils promote VLA-4-dependent B cell antigen presentation and accumulation within the meninges during neuroinflammation‏
670 ‎‡a Author's Non-apoptotic Fas‏
670 ‎‡a Author's Non-apoptotic Fas (CD95) Signaling on T Cells Regulates the Resolution of Th2-Mediated Inflammation‏
670 ‎‡a Author's Protective effector memory CD4 T cells depend on ICOS for survival‏
670 ‎‡a Author's Regulation of myofibroblast differentiation and bleomycin-induced pulmonary fibrosis by adrenomedullin‏
670 ‎‡a Author's RGS3 controls T lymphocyte migration in a model of Th2-mediated airway inflammation‏
670 ‎‡a Author's Self-renewing resident arterial macrophages arise from embryonic CX3CR1(+) precursors and circulating monocytes immediately after birth‏
670 ‎‡a Author's Signaling through FcRγ-associated receptors on dendritic cells drives IL-33-dependent TH2-type responses.‏
670 ‎‡a Author's Thermoneutrality but Not UCP1 Deficiency Suppresses Monocyte Mobilization Into Blood.‏
670 ‎‡a Author's Transcription factor IRF4 drives dendritic cells to promote Th2 differentiation.‏
670 ‎‡a Author's Transcriptome Analysis Reveals Nonfoamy Rather Than Foamy Plaque Macrophages Are Proinflammatory in Atherosclerotic Murine Models‏
909 ‎‡a (scopus) 55499938500‏ ‎‡9 1‏
909 ‎‡a (orcid) 0000000338150891‏ ‎‡9 1‏
919 ‎‡a myocardialbcellsareasubsetofcirculatinglymphocyteswithdelayedtransitthroughtheheart‏ ‎‡A Myocardial B cells are a subset of circulating lymphocytes with delayed transit through the heart‏ ‎‡9 1‏
919 ‎‡a monocyterecruitmentspecificationandfunctioninatherosclerosis‏ ‎‡A Monocyte Recruitment, Specification, and Function in Atherosclerosis‏ ‎‡9 1‏
919 ‎‡a mhcii+residentperitonealandpleuralmacrophagesrelyonirf4fordevelopmentfromcirculatingmonocytes‏ ‎‡A MHC II+ resident peritoneal and pleural macrophages rely on IRF4 for development from circulating monocytes‏ ‎‡9 1‏
919 ‎‡a macrophagebiologyclassificationandphenotypeincardiovasculardiseasejaccmacrophageincvdseriespart1‏ ‎‡A Macrophage Biology, Classification, and Phenotype in Cardiovascular Disease: JACC Macrophage in CVD Series (Part 1)‏ ‎‡9 1‏
919 ‎‡a limitedproliferationcapacityofaorticintimaresidentmacrophagesrequiresmonocyterecruitmentforatheroscleroticplaqueprogression‏ ‎‡A Limited proliferation capacity of aortic intima resident macrophages requires monocyte recruitment for atherosclerotic plaque progression‏ ‎‡9 1‏
919 ‎‡a limitedmacrophagepositionaldynamicsinprogressingorregressingmurineatheroscleroticplaquesbriefreport‏ ‎‡A Limited Macrophage Positional Dynamics in Progressing or Regressing Murine Atherosclerotic Plaques-Brief Report‏ ‎‡9 1‏
919 ‎‡a 33dependentinductionofallergiclunginflammationbyfcγriiisignaling‏ ‎‡A IL-33-dependent induction of allergic lung inflammation by FcγRIII signaling.‏ ‎‡9 1‏
919 ‎‡a icosprotectsagainstmortalityfromacutelunginjurythroughactivationofil5+ilc2s‏ ‎‡A ICOS protects against mortality from acute lung injury through activation of IL-5+ ILC2s‏ ‎‡9 1‏
919 ‎‡a icosexpressinglymphocytespromoteresolutionofcd8mediatedlunginjuryinamousemodeloflungrejection‏ ‎‡A ICOS-expressing lymphocytes promote resolution of CD8-mediated lung injury in a mouse model of lung rejection‏ ‎‡9 1‏
919 ‎‡a homeostaticcontrolofinnatelunginflammationbyvicisyndromegeneepg5andadditionalautophagygenespromotesinfluenzapathogenesis‏ ‎‡A Homeostatic Control of Innate Lung Inflammation by Vici Syndrome Gene Epg5 and Additional Autophagy Genes Promotes Influenza Pathogenesis‏ ‎‡9 1‏
919 ‎‡a fasligandexpressionontcellsissufficienttopreventprolongedairwayinflammationinamurinemodelofasthma‏ ‎‡A Fas ligand expression on T cells is sufficient to prevent prolonged airway inflammation in a murine model of asthma.‏ ‎‡9 1‏
919 ‎‡a cytokinecircuitsincardiovasculardisease‏ ‎‡A Cytokine Circuits in Cardiovascular Disease‏ ‎‡9 1‏
919 ‎‡a contributionofmetabolicdiseasetobonefragilityinmagp1deficientmice‏ ‎‡A Contribution of metabolic disease to bone fragility in MAGP1-deficient mice.‏ ‎‡9 1‏
919 ‎‡a cloningandexpressionofazebrafish5ht2creceptorgene‏ ‎‡A Cloning and expression of a zebrafish 5-HT(2C) receptor gene.‏ ‎‡9 1‏
919 ‎‡a cloningandexpressionofazebrafish5ht‏ ‎‡A Cloning and expression of a zebrafish 5-HT‏ ‎‡9 1‏
919 ‎‡a ccr7andirf4dependentdendriticcellsregulatelymphaticcollectingvesselpermeability‏ ‎‡A CCR7 and IRF4-dependent dendritic cells regulate lymphatic collecting vessel permeability‏ ‎‡9 1‏
919 ‎‡a brownadiposetissuemonocytessupporttissueexpansion‏ ‎‡A Brown adipose tissue monocytes support tissue expansion‏ ‎‡9 1‏
919 ‎‡a bcellmediatedantigenpresentationthroughmhcclass2isdispensableforatherosclerosisprogression‏ ‎‡A B Cell-Mediated Antigen Presentation through MHC Class II Is Dispensable for Atherosclerosis Progression‏ ‎‡9 1‏
919 ‎‡a allergenexposureinlymphopenicfasdeficientmiceresultsinpersistenteosinophiliaduetodefectsinresolutionofinflammation‏ ‎‡A Allergen Exposure in Lymphopenic Fas-Deficient Mice Results in Persistent Eosinophilia Due to Defects in Resolution of Inflammation‏ ‎‡9 1‏
919 ‎‡a stromalnichedefinedbyexpressionofthetranscriptionfactorwt1mediatesprogrammingandhomeostasisofcavityresidentmacrophages‏ ‎‡A A Stromal Niche Defined by Expression of the Transcription Factor WT1 Mediates Programming and Homeostasis of Cavity-Resident Macrophages‏ ‎‡9 1‏
919 ‎‡a polecatsviewofpatrollingmonocytes‏ ‎‡A A Polecat's View of Patrolling Monocytes.‏ ‎‡9 1‏
919 ‎‡a transcriptomeanalysisrevealsnonfoamyratherthanfoamyplaquemacrophagesareproinflammatoryinatheroscleroticmurinemodels‏ ‎‡A Transcriptome Analysis Reveals Nonfoamy Rather Than Foamy Plaque Macrophages Are Proinflammatory in Atherosclerotic Murine Models‏ ‎‡9 1‏
919 ‎‡a transcriptionfactorirf4drivesdendriticcellstopromoteth2differentiation‏ ‎‡A Transcription factor IRF4 drives dendritic cells to promote Th2 differentiation.‏ ‎‡9 1‏
919 ‎‡a thermoneutralitybutnotucp1deficiencysuppressesmonocytemobilizationintoblood‏ ‎‡A Thermoneutrality but Not UCP1 Deficiency Suppresses Monocyte Mobilization Into Blood.‏ ‎‡9 1‏
919 ‎‡a signalingthroughfcrγassociatedreceptorsondendriticcellsdrivesil33dependentth2typeresponses‏ ‎‡A Signaling through FcRγ-associated receptors on dendritic cells drives IL-33-dependent TH2-type responses.‏ ‎‡9 1‏
919 ‎‡a selfrenewingresidentarterialmacrophagesarisefromembryoniccx3cr1+precursorsandcirculatingmonocytesimmediatelyafterbirth‏ ‎‡A Self-renewing resident arterial macrophages arise from embryonic CX3CR1(+) precursors and circulating monocytes immediately after birth‏ ‎‡9 1‏
919 ‎‡a rgs3controlstlymphocytemigrationinamodelofth2mediatedairwayinflammation‏ ‎‡A RGS3 controls T lymphocyte migration in a model of Th2-mediated airway inflammation‏ ‎‡9 1‏
919 ‎‡a regulationofmyofibroblastdifferentiationandbleomycininducedpulmonaryfibrosisbyadrenomedullin‏ ‎‡A Regulation of myofibroblast differentiation and bleomycin-induced pulmonary fibrosis by adrenomedullin‏ ‎‡9 1‏
919 ‎‡a protectiveeffectormemorycd4tcellsdependonicosforsurvival‏ ‎‡A Protective effector memory CD4 T cells depend on ICOS for survival‏ ‎‡9 1‏
919 ‎‡a nonapoptoticfascd95signalingontcellsregulatestheresolutionofth2mediatedinflammation‏ ‎‡A Non-apoptotic Fas (CD95) Signaling on T Cells Regulates the Resolution of Th2-Mediated Inflammation‏ ‎‡9 1‏
919 ‎‡a nonapoptoticfas‏ ‎‡A Non-apoptotic Fas‏ ‎‡9 1‏
919 ‎‡a neutrophilspromotevla4dependentbcellantigenpresentationandaccumulationwithinthemeningesduringneuroinflammation‏ ‎‡A Neutrophils promote VLA-4-dependent B cell antigen presentation and accumulation within the meninges during neuroinflammation‏ ‎‡9 1‏
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