Search
Leader | 00000nz a2200037n 45 0 | ||
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001 | WKP|Q57140999 (VIAF cluster) (Authority/Source Record) | ||
003 | WKP | ||
005 | 20241221010844.0 | ||
008 | 241221nneanz||abbn n and d | ||
035 | ‡a (WKP)Q57140999 | ||
024 | ‡a 0000-0002-9864-2268 ‡2 orcid | ||
035 | ‡a (OCoLC)Q57140999 | ||
100 | 0 | ‡a Jonathan D. Katz ‡c researcher ‡9 en | |
375 | ‡a 1 ‡2 iso5218 | ||
400 | 0 | ‡a Jonathan D. Katz ‡c wetenschapper ‡9 nl | |
670 | ‡a Author's Bim/Bcl-2 balance is critical for maintaining naive and memory T cell homeostasis | ||
670 | ‡a Author's Breaking T cell tolerance to beta cell antigens by merocytic dendritic cells | ||
670 | ‡a Author's CD8(+) T cells specific for beta cells encounter their cognate antigens in the islets of NOD mice. | ||
670 | ‡a Author's Cutting edge: merocytic dendritic cells break T cell tolerance to beta cell antigens in nonobese diabetic mouse diabetes. | ||
670 | ‡a Author's Dying to protect: cell death and the control of T-cell homeostasis. | ||
670 | ‡a Author's Dynamic interaction between T cell-mediated β-cell damage and β-cell repair in the run up to autoimmune diabetes of the NOD mouse | ||
670 | ‡a Author's Eliminating encephalitogenic T cells without undermining protective immunity | ||
670 | ‡a Author's Etoposide selectively ablates activated T cells to control the immunoregulatory disorder hemophagocytic lymphohistiocytosis. | ||
670 | ‡a Author's Extending Remission and Reversing New-Onset Type 1 Diabetes by Targeted Ablation of Autoreactive T Cells | ||
670 | ‡a Author's Following a diabetogenic T cell from genesis through pathogenesis | ||
670 | ‡a Author's Genetic control of diabetes progression | ||
670 | ‡a Author's IFN-γ receptor deficiency prevents diabetes induction by diabetogenic CD4+, but not CD8+, T cells | ||
670 | ‡a Author's IL-10 deficiency does not inhibit insulitis and accelerates cyclophosphamide-induced diabetes in the nonobese diabetic mouse. | ||
670 | ‡a Author's In autoimmune diabetes the transition from benign to pernicious insulitis requires an islet cell response to tumor necrosis factor alpha. | ||
670 | ‡a Author's Interferon-gamma impacts at multiple points during the progression of autoimmune diabetes | ||
670 | ‡a Author's Islet-specific T-cell clones from nonobese diabetic mice express heterogeneous T-cell receptors. | ||
670 | ‡a Author's Major histocompatibility complex class I molecules are required for the development of insulitis in non-obese diabetic mice | ||
670 | ‡a Author's Major Histocompatibility Complex Class II Molecules Can Protect from Diabetes by Positively Selecting T Cells with Additional Specificities | ||
670 | ‡a Author's Manipulating DNA damage-response signaling for the treatment of immune-mediated diseases | ||
670 | ‡a Author's Mechanism of NK activation by OK-432 | ||
670 | ‡a Author's Mechanism of NK activation by OK-432 (Streptococcus pyogenes). I. Spontaneous release of NKCF and augmentation of NKCF production following stimulation with NK target cells | ||
670 | ‡a Author's Mechanism of T-dependent cytotoxicity: role of papain-sensitive non class I MHC target molecules and expression of target antigen for cytotoxicity | ||
670 | ‡a Author's Microarray and comparative genomics-based identification of genes and gene regulatory regions of the mouse immune system | ||
670 | ‡a Author's NKT cells and IFN-gamma establish the regulatory environment for the control of diabetogenic T cells in the nonobese diabetic mouse | ||
670 | ‡a Author's Regulation of autoimmune diabetes by non-islet-specific T cells - a role for the glucocorticoid-induced TNF receptor | ||
670 | ‡a Author's Reversal of New-Onset Type 1 Diabetes With an Agonistic TLR4/MD-2 Monoclonal Antibody. | ||
670 | ‡a Author's T cells instruct myeloid cells to produce inflammasome-independent IL-1β and cause autoimmunity | ||
670 | ‡a Author's T Helper 2 (Th2) T Cells Induce Acute Pancreatitis and Diabetes in Immune-compromised Nonobese Diabetic (NOD) Mice | ||
670 | ‡a Author's T helper cell subsets in insulin-dependent diabetes | ||
670 | ‡a Author's The countervailing actions of myeloid and plasmacytoid dendritic cells control autoimmune diabetes in the nonobese diabetic mouse. | ||
670 | ‡a Author's The SJL/J T cell response to both spontaneous and transplantable syngeneic reticulum cell sarcoma is mediated predominantly by the V beta 17a+ T cell clonotype. | ||
909 | ‡a (orcid) 0000000298642268 ‡9 1 | ||
919 | ‡a reversalofnewonsettype1diabeteswithanagonistictlr415002monoclonalantibody ‡A Reversal of New-Onset Type 1 Diabetes With an Agonistic TLR4/MD-2 Monoclonal Antibody. ‡9 1 | ||
919 | ‡a regulationofautoimmunediabetesbynonisletspecifictcellsarolefortheglucocorticoidinducedtnfreceptor ‡A Regulation of autoimmune diabetes by non-islet-specific T cells - a role for the glucocorticoid-induced TNF receptor ‡9 1 | ||
919 | ‡a nktcellsandifngammaestablishtheregulatoryenvironmentforthecontrolofdiabetogenictcellsinthenonobesediabeticmouse ‡A NKT cells and IFN-gamma establish the regulatory environment for the control of diabetogenic T cells in the nonobese diabetic mouse ‡9 1 | ||
919 | ‡a microarrayandcomparativegenomicsbasedidentificationofgenesandgeneregulatoryregionsofthemouseimmunesystem ‡A Microarray and comparative genomics-based identification of genes and gene regulatory regions of the mouse immune system ‡9 1 | ||
919 | ‡a mechanismoftdependentcytotoxicityroleofpapainsensitivenonclass1mhctargetmoleculesandexpressionoftargetantigenforcytotoxicity ‡A Mechanism of T-dependent cytotoxicity: role of papain-sensitive non class I MHC target molecules and expression of target antigen for cytotoxicity ‡9 1 | ||
919 | ‡a mechanismofnkactivationbyok432streptococcuspyogenes1spontaneousreleaseofnkcfandaugmentationofnkcfproductionfollowingstimulationwithnktargetcells ‡A Mechanism of NK activation by OK-432 (Streptococcus pyogenes). I. Spontaneous release of NKCF and augmentation of NKCF production following stimulation with NK target cells ‡9 1 | ||
919 | ‡a mechanismofnkactivationbyok432 ‡A Mechanism of NK activation by OK-432 ‡9 1 | ||
919 | ‡a manipulatingdnadamageresponsesignalingforthetreatmentofimmunemediateddiseases ‡A Manipulating DNA damage-response signaling for the treatment of immune-mediated diseases ‡9 1 | ||
919 | ‡a majorhistocompatibilitycomplexclass2moleculescanprotectfromdiabetesbypositivelyselectingtcellswithadditionalspecificities ‡A Major Histocompatibility Complex Class II Molecules Can Protect from Diabetes by Positively Selecting T Cells with Additional Specificities ‡9 1 | ||
919 | ‡a majorhistocompatibilitycomplexclass1moleculesarerequiredforthedevelopmentofinsulitisinnonobesediabeticmice ‡A Major histocompatibility complex class I molecules are required for the development of insulitis in non-obese diabetic mice ‡9 1 | ||
919 | ‡a isletspecifictcellclonesfromnonobesediabeticmiceexpressheterogeneoustcellreceptors ‡A Islet-specific T-cell clones from nonobese diabetic mice express heterogeneous T-cell receptors. ‡9 1 | ||
919 | ‡a interferongammaimpactsatmultiplepointsduringtheprogressionofautoimmunediabetes ‡A Interferon-gamma impacts at multiple points during the progression of autoimmune diabetes ‡9 1 | ||
919 | ‡a inautoimmunediabetesthetransitionfrombenigntoperniciousinsulitisrequiresanisletcellresponsetotumornecrosisfactoralpha ‡A In autoimmune diabetes the transition from benign to pernicious insulitis requires an islet cell response to tumor necrosis factor alpha. ‡9 1 | ||
919 | ‡a 10deficiencydoesnotinhibitinsulitisandacceleratescyclophosphamideinduceddiabetesinthenonobesediabeticmouse ‡A IL-10 deficiency does not inhibit insulitis and accelerates cyclophosphamide-induced diabetes in the nonobese diabetic mouse. ‡9 1 | ||
919 | ‡a ifnγreceptordeficiencypreventsdiabetesinductionbydiabetogeniccd4+butnotcd8+tcells ‡A IFN-γ receptor deficiency prevents diabetes induction by diabetogenic CD4+, but not CD8+, T cells ‡9 1 | ||
919 | ‡a geneticcontrolofdiabetesprogression ‡A Genetic control of diabetes progression ‡9 1 | ||
919 | ‡a followingadiabetogenictcellfromgenesisthroughpathogenesis ‡A Following a diabetogenic T cell from genesis through pathogenesis ‡9 1 | ||
919 | ‡a extendingremissionandreversingnewonsettype1diabetesbytargetedablationofautoreactivetcells ‡A Extending Remission and Reversing New-Onset Type 1 Diabetes by Targeted Ablation of Autoreactive T Cells ‡9 1 | ||
919 | ‡a etoposideselectivelyablatesactivatedtcellstocontroltheimmunoregulatorydisorderhemophagocyticlymphohistiocytosis ‡A Etoposide selectively ablates activated T cells to control the immunoregulatory disorder hemophagocytic lymphohistiocytosis. ‡9 1 | ||
919 | ‡a eliminatingencephalitogenictcellswithoutunderminingprotectiveimmunity ‡A Eliminating encephalitogenic T cells without undermining protective immunity ‡9 1 | ||
919 | ‡a dynamicinteractionbetweentcellmediatedβcelldamageandβcellrepairintherunuptoautoimmunediabetesofthenodmouse ‡A Dynamic interaction between T cell-mediated β-cell damage and β-cell repair in the run up to autoimmune diabetes of the NOD mouse ‡9 1 | ||
919 | ‡a dyingtoprotectcelldeathandthecontroloftcellhomeostasis ‡A Dying to protect: cell death and the control of T-cell homeostasis. ‡9 1 | ||
919 | ‡a cuttingedgemerocyticdendriticcellsbreaktcelltolerancetobetacellantigensinnonobesediabeticmousediabetes ‡A Cutting edge: merocytic dendritic cells break T cell tolerance to beta cell antigens in nonobese diabetic mouse diabetes. ‡9 1 | ||
919 | ‡a cd8+tcellsspecificforbetacellsencountertheircognateantigensintheisletsofnodmice ‡A CD8(+) T cells specific for beta cells encounter their cognate antigens in the islets of NOD mice. ‡9 1 | ||
919 | ‡a breakingtcelltolerancetobetacellantigensbymerocyticdendriticcells ‡A Breaking T cell tolerance to beta cell antigens by merocytic dendritic cells ‡9 1 | ||
919 | ‡a bimbcl2balanceiscriticalformaintainingnaiveandmemorytcellhomeostasis ‡A Bim/Bcl-2 balance is critical for maintaining naive and memory T cell homeostasis ‡9 1 | ||
919 | ‡a sjljtcellresponsetobothspontaneousandtransplantablesyngeneicreticulumcellsarcomaismediatedpredominantlybythe5beta17a+tcellclonotype ‡A The SJL/J T cell response to both spontaneous and transplantable syngeneic reticulum cell sarcoma is mediated predominantly by the V beta 17a+ T cell clonotype. ‡9 1 | ||
919 | ‡a countervailingactionsofmyeloidandplasmacytoiddendriticcellscontrolautoimmunediabetesinthenonobesediabeticmouse ‡A The countervailing actions of myeloid and plasmacytoid dendritic cells control autoimmune diabetes in the nonobese diabetic mouse. ‡9 1 | ||
919 | ‡a thelpercellsubsetsininsulindependentdiabetes ‡A T helper cell subsets in insulin-dependent diabetes ‡9 1 | ||
919 | ‡a thelper2th2tcellsinduceacutepancreatitisanddiabetesinimmunecompromisednonobesediabeticnodmice ‡A T Helper 2 (Th2) T Cells Induce Acute Pancreatitis and Diabetes in Immune-compromised Nonobese Diabetic (NOD) Mice ‡9 1 | ||
919 | ‡a tcellsinstructmyeloidcellstoproduceinflammasomeindependentil1βandcauseautoimmunity ‡A T cells instruct myeloid cells to produce inflammasome-independent IL-1β and cause autoimmunity ‡9 1 | ||
946 | ‡a b ‡9 1 | ||
996 | ‡2 J9U|987011561231905171 | ||
996 | ‡2 NUKAT|n 2008088475 | ||
996 | ‡2 SUDOC|179731394 | ||
996 | ‡2 J9U|987007294130405171 | ||
996 | ‡2 ISNI|0000000074276254 | ||
996 | ‡2 ISNI|0000000054628400 | ||
996 | ‡2 J9U|987007263776105171 | ||
996 | ‡2 SIMACOB|239042659 | ||
996 | ‡2 DNB|1169835678 | ||
996 | ‡2 LC|no2010011562 | ||
996 | ‡2 ISNI|000000007883433X | ||
996 | ‡2 BNF|13564279 | ||
996 | ‡2 NII|DA00601871 | ||
996 | ‡2 BIBSYS|98005205 | ||
996 | ‡2 J9U|987007296969405171 | ||
996 | ‡2 BNF|13499053 | ||
996 | ‡2 ISNI|0000000122132205 | ||
996 | ‡2 LC|n 83056037 | ||
996 | ‡2 BIBSYS|90160114 | ||
996 | ‡2 DNB|1062578090 | ||
996 | ‡2 LC|no 97063592 | ||
996 | ‡2 J9U|987007443424405171 | ||
996 | ‡2 NLR|RU NLR AUTH 7724475 | ||
996 | ‡2 LC|n 2016017377 | ||
996 | ‡2 BIBSYS|90930005 | ||
996 | ‡2 B2Q|0001062012 | ||
996 | ‡2 NUKAT|n 96600152 | ||
996 | ‡2 NUKAT|n 2017048398 | ||
996 | ‡2 PTBNP|1564982 | ||
996 | ‡2 NTA|072064692 | ||
996 | ‡2 BIBSYS|10019133 | ||
996 | ‡2 NII|DA02967852 | ||
996 | ‡2 NII|DA16063054 | ||
996 | ‡2 DNB|120064686X | ||
996 | ‡2 DNB|1089169728 | ||
996 | ‡2 NTA|260836702 | ||
996 | ‡2 LC|n 2001109116 | ||
996 | ‡2 BNE|XX1112454 | ||
996 | ‡2 J9U|987007390678005171 | ||
996 | ‡2 DNB|18848342X | ||
996 | ‡2 ISNI|0000000029683096 | ||
996 | ‡2 SELIBR|311450 | ||
996 | ‡2 BLBNB|000429591 | ||
996 | ‡2 SUDOC|183423127 | ||
996 | ‡2 DNB|1019873922 | ||
996 | ‡2 LC|n 92087673 | ||
996 | ‡2 DNB|137507828 | ||
996 | ‡2 NDL|01164768 | ||
996 | ‡2 NUKAT|n 98042819 | ||
996 | ‡2 J9U|987007302436105171 | ||
996 | ‡2 DNB|132467658 | ||
996 | ‡2 ISNI|0000000394856637 | ||
996 | ‡2 CAOONL|ncf11439025 | ||
996 | ‡2 SUDOC|035721367 | ||
996 | ‡2 LC|n 2012063211 | ||
996 | ‡2 NUKAT|n 2008111120 | ||
996 | ‡2 RERO|A003444580 | ||
996 | ‡2 LC|n 90685257 | ||
996 | ‡2 BNF|16204722 | ||
996 | ‡2 SIMACOB|24912227 | ||
996 | ‡2 LC|nr 90008587 | ||
996 | ‡2 LC|no2019062410 | ||
996 | ‡2 NTA|097903019 | ||
996 | ‡2 BAV|495_323821 | ||
996 | ‡2 ISNI|0000000082854149 | ||
996 | ‡2 CAOONL|ncf11276889 | ||
996 | ‡2 BIBSYS|90669665 | ||
996 | ‡2 LC|n 2010074080 | ||
996 | ‡2 LC|n 87114122 | ||
996 | ‡2 CAOONL|ncf11879454 | ||
996 | ‡2 DNB|1201126878 | ||
996 | ‡2 LC|n 2016038157 | ||
996 | ‡2 LC|n 2014013163 | ||
996 | ‡2 ISNI|000000003498677X | ||
996 | ‡2 ISNI|0000000079073739 | ||
996 | ‡2 NUKAT|n 02032832 | ||
996 | ‡2 SUDOC|086012096 | ||
996 | ‡2 BIBSYS|13008076 | ||
996 | ‡2 SIMACOB|306066531 | ||
996 | ‡2 BIBSYS|4022960 | ||
996 | ‡2 BIBSYS|8074590 | ||
996 | ‡2 LC|no2010169493 | ||
996 | ‡2 DNB|132990652 | ||
996 | ‡2 DNB|133268101 | ||
996 | ‡2 ISNI|0000000110607680 | ||
996 | ‡2 SZ|133268101 | ||
996 | ‡2 ISNI|0000000135955622 | ||
996 | ‡2 SUDOC|199466041 | ||
996 | ‡2 ISNI|0000000110778009 | ||
996 | ‡2 NKC|stk2007415724 | ||
996 | ‡2 PLWABN|9812826143905606 | ||
996 | ‡2 BIBSYS|4001764 | ||
996 | ‡2 ISNI|0000000114497861 | ||
996 | ‡2 LC|no2006111951 | ||
996 | ‡2 NII|DA19961613 | ||
996 | ‡2 CAOONL|ncf10158543 | ||
996 | ‡2 ISNI|0000000114777345 | ||
996 | ‡2 ISNI|0000000430345181 | ||
996 | ‡2 ISNI|0000000042364436 | ||
996 | ‡2 NUKAT|n 2013182974 | ||
996 | ‡2 CAOONL|ncf12120134 | ||
996 | ‡2 J9U|987007422764705171 | ||
996 | ‡2 NTA|074778986 | ||
996 | ‡2 CAOONL|ncf11047724 | ||
996 | ‡2 ISNI|0000000041138904 | ||
996 | ‡2 SUDOC|035196750 | ||
996 | ‡2 BIBSYS|2027426 | ||
996 | ‡2 J9U|987007460900605171 | ||
996 | ‡2 LC|nb2007014894 | ||
996 | ‡2 SUDOC|249792117 | ||
996 | ‡2 CAOONL|ncf11709232 | ||
996 | ‡2 NII|DA01310229 | ||
996 | ‡2 ISNI|0000000366448054 | ||
996 | ‡2 NKC|jo20010090073 | ||
996 | ‡2 NUKAT|n 2018093239 | ||
996 | ‡2 NTA|071847855 | ||
996 | ‡2 J9U|987007375462505171 | ||
996 | ‡2 DNB|1157169260 | ||
996 | ‡2 NKC|xx0315816 | ||
996 | ‡2 ISNI|0000000105206815 | ||
996 | ‡2 LC|n 50044659 | ||
996 | ‡2 J9U|987007423353305171 | ||
996 | ‡2 BNF|15082161 | ||
996 | ‡2 BNE|XX5668031 | ||
996 | ‡2 BLBNB|000255101 | ||
996 | ‡2 ISNI|0000000107634987 | ||
996 | ‡2 LC|no2009178555 | ||
996 | ‡2 RERO|A013877658 | ||
996 | ‡2 RERO|A013877659 | ||
996 | ‡2 LC|n 2016003740 | ||
996 | ‡2 ISNI|0000000381620684 | ||
996 | ‡2 PLWABN|9812484443905606 | ||
996 | ‡2 CAOONL|ncf10962775 | ||
996 | ‡2 RERO|A003444581 | ||
996 | ‡2 CAOONL|ncf12115416 | ||
996 | ‡2 RERO|A003444582 | ||
996 | ‡2 J9U|987007263772505171 | ||
996 | ‡2 CAOONL|ncf10072930 | ||
996 | ‡2 CAOONL|ncf11167629 | ||
996 | ‡2 NUKAT|n 2010113260 | ||
996 | ‡2 CAOONL|ncf10872810 | ||
996 | ‡2 DNB|137008953 | ||
996 | ‡2 LC|nr 97018173 | ||
996 | ‡2 LC|nb 98016981 | ||
996 | ‡2 SUDOC|119075628 | ||
996 | ‡2 J9U|987007388764505171 | ||
996 | ‡2 DNB|1053434529 | ||
996 | ‡2 SUDOC|060400749 | ||
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996 | ‡2 PLWABN|9810661027405606 | ||
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996 | ‡2 PLWABN|9810534191705606 | ||
996 | ‡2 J9U|987007441154605171 | ||
996 | ‡2 LC|n 86839116 | ||
996 | ‡2 NUKAT|n 97022310 | ||
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996 | ‡2 ISNI|0000000040305264 | ||
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996 | ‡2 NDL|00469305 | ||
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996 | ‡2 NTA|305841858 | ||
996 | ‡2 J9U|987007271820705171 | ||
996 | ‡2 LC|n 85296374 | ||
996 | ‡2 NYNYRILM|207242 | ||
996 | ‡2 J9U|987007460150205171 | ||
996 | ‡2 J9U|987007327163705171 | ||
996 | ‡2 DNB|101736284X | ||
996 | ‡2 NKC|mzk2009502227 | ||
996 | ‡2 BNF|12298538 | ||
996 | ‡2 ISNI|0000000071441293 | ||
996 | ‡2 BIBSYS|2039087 | ||
996 | ‡2 RERO|A013874168 | ||
996 | ‡2 KRNLK|KAC200507721 | ||
996 | ‡2 SUDOC|082969302 | ||
996 | ‡2 BNF|12691161 | ||
996 | ‡2 LC|n 82119230 | ||
996 | ‡2 RERO|A003444579 | ||
996 | ‡2 BNF|17006773 | ||
996 | ‡2 NKC|jn20010601256 | ||
996 | ‡2 LC|n 90600823 | ||
996 | ‡2 LC|no2019011430 | ||
996 | ‡2 SUDOC|131136348 | ||
996 | ‡2 LC|no 98119018 | ||
996 | ‡2 DNB|1254692207 | ||
996 | ‡2 NII|DA0995702X | ||
996 | ‡2 CAOONL|ncf13761772 | ||
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996 | ‡2 J9U|987007294109905171 | ||
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996 | ‡2 DBC|87097947235359 | ||
996 | ‡2 NTA|323975380 | ||
996 | ‡2 SUDOC|124880940 | ||
996 | ‡2 BNF|13477136 | ||
997 | ‡a 0 0 lived 0 0 ‡9 1 |