VIAF

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Leader 00000nz a2200037n 45 0
001 WKP|Q57140999 (VIAF cluster) (Authority/Source Record)
003 WKP
005 20241221010844.0
008 241221nneanz||abbn n and d
035 ‎‡a (WKP)Q57140999‏
024 ‎‡a 0000-0002-9864-2268‏ ‎‡2 orcid‏
035 ‎‡a (OCoLC)Q57140999‏
100 0 ‎‡a Jonathan D. Katz‏ ‎‡c researcher‏ ‎‡9 en‏
375 ‎‡a 1‏ ‎‡2 iso5218‏
400 0 ‎‡a Jonathan D. Katz‏ ‎‡c wetenschapper‏ ‎‡9 nl‏
670 ‎‡a Author's Bim/Bcl-2 balance is critical for maintaining naive and memory T cell homeostasis‏
670 ‎‡a Author's Breaking T cell tolerance to beta cell antigens by merocytic dendritic cells‏
670 ‎‡a Author's CD8(+) T cells specific for beta cells encounter their cognate antigens in the islets of NOD mice.‏
670 ‎‡a Author's Cutting edge: merocytic dendritic cells break T cell tolerance to beta cell antigens in nonobese diabetic mouse diabetes.‏
670 ‎‡a Author's Dying to protect: cell death and the control of T-cell homeostasis.‏
670 ‎‡a Author's Dynamic interaction between T cell-mediated β-cell damage and β-cell repair in the run up to autoimmune diabetes of the NOD mouse‏
670 ‎‡a Author's Eliminating encephalitogenic T cells without undermining protective immunity‏
670 ‎‡a Author's Etoposide selectively ablates activated T cells to control the immunoregulatory disorder hemophagocytic lymphohistiocytosis.‏
670 ‎‡a Author's Extending Remission and Reversing New-Onset Type 1 Diabetes by Targeted Ablation of Autoreactive T Cells‏
670 ‎‡a Author's Following a diabetogenic T cell from genesis through pathogenesis‏
670 ‎‡a Author's Genetic control of diabetes progression‏
670 ‎‡a Author's IFN-γ receptor deficiency prevents diabetes induction by diabetogenic CD4+, but not CD8+, T cells‏
670 ‎‡a Author's IL-10 deficiency does not inhibit insulitis and accelerates cyclophosphamide-induced diabetes in the nonobese diabetic mouse.‏
670 ‎‡a Author's In autoimmune diabetes the transition from benign to pernicious insulitis requires an islet cell response to tumor necrosis factor alpha.‏
670 ‎‡a Author's Interferon-gamma impacts at multiple points during the progression of autoimmune diabetes‏
670 ‎‡a Author's Islet-specific T-cell clones from nonobese diabetic mice express heterogeneous T-cell receptors.‏
670 ‎‡a Author's Major histocompatibility complex class I molecules are required for the development of insulitis in non-obese diabetic mice‏
670 ‎‡a Author's Major Histocompatibility Complex Class II Molecules Can Protect from Diabetes by Positively Selecting T Cells with Additional Specificities‏
670 ‎‡a Author's Manipulating DNA damage-response signaling for the treatment of immune-mediated diseases‏
670 ‎‡a Author's Mechanism of NK activation by OK-432‏
670 ‎‡a Author's Mechanism of NK activation by OK-432 (Streptococcus pyogenes). I. Spontaneous release of NKCF and augmentation of NKCF production following stimulation with NK target cells‏
670 ‎‡a Author's Mechanism of T-dependent cytotoxicity: role of papain-sensitive non class I MHC target molecules and expression of target antigen for cytotoxicity‏
670 ‎‡a Author's Microarray and comparative genomics-based identification of genes and gene regulatory regions of the mouse immune system‏
670 ‎‡a Author's NKT cells and IFN-gamma establish the regulatory environment for the control of diabetogenic T cells in the nonobese diabetic mouse‏
670 ‎‡a Author's Regulation of autoimmune diabetes by non-islet-specific T cells - a role for the glucocorticoid-induced TNF receptor‏
670 ‎‡a Author's Reversal of New-Onset Type 1 Diabetes With an Agonistic TLR4/MD-2 Monoclonal Antibody.‏
670 ‎‡a Author's T cells instruct myeloid cells to produce inflammasome-independent IL-1β and cause autoimmunity‏
670 ‎‡a Author's T Helper 2 (Th2) T Cells Induce Acute Pancreatitis and Diabetes in Immune-compromised Nonobese Diabetic (NOD) Mice‏
670 ‎‡a Author's T helper cell subsets in insulin-dependent diabetes‏
670 ‎‡a Author's The countervailing actions of myeloid and plasmacytoid dendritic cells control autoimmune diabetes in the nonobese diabetic mouse.‏
670 ‎‡a Author's The SJL/J T cell response to both spontaneous and transplantable syngeneic reticulum cell sarcoma is mediated predominantly by the V beta 17a+ T cell clonotype.‏
909 ‎‡a (orcid) 0000000298642268‏ ‎‡9 1‏
919 ‎‡a reversalofnewonsettype1diabeteswithanagonistictlr415002monoclonalantibody‏ ‎‡A Reversal of New-Onset Type 1 Diabetes With an Agonistic TLR4/MD-2 Monoclonal Antibody.‏ ‎‡9 1‏
919 ‎‡a regulationofautoimmunediabetesbynonisletspecifictcellsarolefortheglucocorticoidinducedtnfreceptor‏ ‎‡A Regulation of autoimmune diabetes by non-islet-specific T cells - a role for the glucocorticoid-induced TNF receptor‏ ‎‡9 1‏
919 ‎‡a nktcellsandifngammaestablishtheregulatoryenvironmentforthecontrolofdiabetogenictcellsinthenonobesediabeticmouse‏ ‎‡A NKT cells and IFN-gamma establish the regulatory environment for the control of diabetogenic T cells in the nonobese diabetic mouse‏ ‎‡9 1‏
919 ‎‡a microarrayandcomparativegenomicsbasedidentificationofgenesandgeneregulatoryregionsofthemouseimmunesystem‏ ‎‡A Microarray and comparative genomics-based identification of genes and gene regulatory regions of the mouse immune system‏ ‎‡9 1‏
919 ‎‡a mechanismoftdependentcytotoxicityroleofpapainsensitivenonclass1mhctargetmoleculesandexpressionoftargetantigenforcytotoxicity‏ ‎‡A Mechanism of T-dependent cytotoxicity: role of papain-sensitive non class I MHC target molecules and expression of target antigen for cytotoxicity‏ ‎‡9 1‏
919 ‎‡a mechanismofnkactivationbyok432streptococcuspyogenes1spontaneousreleaseofnkcfandaugmentationofnkcfproductionfollowingstimulationwithnktargetcells‏ ‎‡A Mechanism of NK activation by OK-432 (Streptococcus pyogenes). I. Spontaneous release of NKCF and augmentation of NKCF production following stimulation with NK target cells‏ ‎‡9 1‏
919 ‎‡a mechanismofnkactivationbyok432‏ ‎‡A Mechanism of NK activation by OK-432‏ ‎‡9 1‏
919 ‎‡a manipulatingdnadamageresponsesignalingforthetreatmentofimmunemediateddiseases‏ ‎‡A Manipulating DNA damage-response signaling for the treatment of immune-mediated diseases‏ ‎‡9 1‏
919 ‎‡a majorhistocompatibilitycomplexclass2moleculescanprotectfromdiabetesbypositivelyselectingtcellswithadditionalspecificities‏ ‎‡A Major Histocompatibility Complex Class II Molecules Can Protect from Diabetes by Positively Selecting T Cells with Additional Specificities‏ ‎‡9 1‏
919 ‎‡a majorhistocompatibilitycomplexclass1moleculesarerequiredforthedevelopmentofinsulitisinnonobesediabeticmice‏ ‎‡A Major histocompatibility complex class I molecules are required for the development of insulitis in non-obese diabetic mice‏ ‎‡9 1‏
919 ‎‡a isletspecifictcellclonesfromnonobesediabeticmiceexpressheterogeneoustcellreceptors‏ ‎‡A Islet-specific T-cell clones from nonobese diabetic mice express heterogeneous T-cell receptors.‏ ‎‡9 1‏
919 ‎‡a interferongammaimpactsatmultiplepointsduringtheprogressionofautoimmunediabetes‏ ‎‡A Interferon-gamma impacts at multiple points during the progression of autoimmune diabetes‏ ‎‡9 1‏
919 ‎‡a inautoimmunediabetesthetransitionfrombenigntoperniciousinsulitisrequiresanisletcellresponsetotumornecrosisfactoralpha‏ ‎‡A In autoimmune diabetes the transition from benign to pernicious insulitis requires an islet cell response to tumor necrosis factor alpha.‏ ‎‡9 1‏
919 ‎‡a 10deficiencydoesnotinhibitinsulitisandacceleratescyclophosphamideinduceddiabetesinthenonobesediabeticmouse‏ ‎‡A IL-10 deficiency does not inhibit insulitis and accelerates cyclophosphamide-induced diabetes in the nonobese diabetic mouse.‏ ‎‡9 1‏
919 ‎‡a ifnγreceptordeficiencypreventsdiabetesinductionbydiabetogeniccd4+butnotcd8+tcells‏ ‎‡A IFN-γ receptor deficiency prevents diabetes induction by diabetogenic CD4+, but not CD8+, T cells‏ ‎‡9 1‏
919 ‎‡a geneticcontrolofdiabetesprogression‏ ‎‡A Genetic control of diabetes progression‏ ‎‡9 1‏
919 ‎‡a followingadiabetogenictcellfromgenesisthroughpathogenesis‏ ‎‡A Following a diabetogenic T cell from genesis through pathogenesis‏ ‎‡9 1‏
919 ‎‡a extendingremissionandreversingnewonsettype1diabetesbytargetedablationofautoreactivetcells‏ ‎‡A Extending Remission and Reversing New-Onset Type 1 Diabetes by Targeted Ablation of Autoreactive T Cells‏ ‎‡9 1‏
919 ‎‡a etoposideselectivelyablatesactivatedtcellstocontroltheimmunoregulatorydisorderhemophagocyticlymphohistiocytosis‏ ‎‡A Etoposide selectively ablates activated T cells to control the immunoregulatory disorder hemophagocytic lymphohistiocytosis.‏ ‎‡9 1‏
919 ‎‡a eliminatingencephalitogenictcellswithoutunderminingprotectiveimmunity‏ ‎‡A Eliminating encephalitogenic T cells without undermining protective immunity‏ ‎‡9 1‏
919 ‎‡a dynamicinteractionbetweentcellmediatedβcelldamageandβcellrepairintherunuptoautoimmunediabetesofthenodmouse‏ ‎‡A Dynamic interaction between T cell-mediated β-cell damage and β-cell repair in the run up to autoimmune diabetes of the NOD mouse‏ ‎‡9 1‏
919 ‎‡a dyingtoprotectcelldeathandthecontroloftcellhomeostasis‏ ‎‡A Dying to protect: cell death and the control of T-cell homeostasis.‏ ‎‡9 1‏
919 ‎‡a cuttingedgemerocyticdendriticcellsbreaktcelltolerancetobetacellantigensinnonobesediabeticmousediabetes‏ ‎‡A Cutting edge: merocytic dendritic cells break T cell tolerance to beta cell antigens in nonobese diabetic mouse diabetes.‏ ‎‡9 1‏
919 ‎‡a cd8+tcellsspecificforbetacellsencountertheircognateantigensintheisletsofnodmice‏ ‎‡A CD8(+) T cells specific for beta cells encounter their cognate antigens in the islets of NOD mice.‏ ‎‡9 1‏
919 ‎‡a breakingtcelltolerancetobetacellantigensbymerocyticdendriticcells‏ ‎‡A Breaking T cell tolerance to beta cell antigens by merocytic dendritic cells‏ ‎‡9 1‏
919 ‎‡a bimbcl2balanceiscriticalformaintainingnaiveandmemorytcellhomeostasis‏ ‎‡A Bim/Bcl-2 balance is critical for maintaining naive and memory T cell homeostasis‏ ‎‡9 1‏
919 ‎‡a sjljtcellresponsetobothspontaneousandtransplantablesyngeneicreticulumcellsarcomaismediatedpredominantlybythe5beta17a+tcellclonotype‏ ‎‡A The SJL/J T cell response to both spontaneous and transplantable syngeneic reticulum cell sarcoma is mediated predominantly by the V beta 17a+ T cell clonotype.‏ ‎‡9 1‏
919 ‎‡a countervailingactionsofmyeloidandplasmacytoiddendriticcellscontrolautoimmunediabetesinthenonobesediabeticmouse‏ ‎‡A The countervailing actions of myeloid and plasmacytoid dendritic cells control autoimmune diabetes in the nonobese diabetic mouse.‏ ‎‡9 1‏
919 ‎‡a thelpercellsubsetsininsulindependentdiabetes‏ ‎‡A T helper cell subsets in insulin-dependent diabetes‏ ‎‡9 1‏
919 ‎‡a thelper2th2tcellsinduceacutepancreatitisanddiabetesinimmunecompromisednonobesediabeticnodmice‏ ‎‡A T Helper 2 (Th2) T Cells Induce Acute Pancreatitis and Diabetes in Immune-compromised Nonobese Diabetic (NOD) Mice‏ ‎‡9 1‏
919 ‎‡a tcellsinstructmyeloidcellstoproduceinflammasomeindependentil1βandcauseautoimmunity‏ ‎‡A T cells instruct myeloid cells to produce inflammasome-independent IL-1β and cause autoimmunity‏ ‎‡9 1‏
946 ‎‡a b‏ ‎‡9 1‏
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