VIAF

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Leader 00000nz a2200037n 45 0
001 WKP|Q58670645 (VIAF cluster) (Authority/Source Record)
003 WKP
005 20241221010728.0
008 241221nneanz||abbn n and d
035 ‎‡a (WKP)Q58670645‏
024 ‎‡a 0000-0001-5109-2062‏ ‎‡2 orcid‏
035 ‎‡a (OCoLC)Q58670645‏
100 0 ‎‡a Marco Matos‏ ‎‡c researcher ORCID ID = 0000-0001-5109-2062‏ ‎‡9 en‏
375 ‎‡a 1‏ ‎‡2 iso5218‏
400 0 ‎‡a Marco Matos‏ ‎‡c wetenschapper‏ ‎‡9 nl‏
670 ‎‡a Author's Adenosine A2A receptors modulate glutamate uptake in cultured astrocytes and gliosomes.‏
670 ‎‡a Author's Amyloid-beta peptide decreases glutamate uptake in cultured astrocytes: involvement of oxidative stress and mitogen-activated protein kinase cascades‏
670 ‎‡a Author's Antagonistic interaction between adenosine A2A receptors and Na+/K+-ATPase-α2 controlling glutamate uptake in astrocytes‏
670 ‎‡a Author's Astrocytes detect and upregulate transmission at inhibitory synapses of somatostatin interneurons onto pyramidal cells‏
670 ‎‡a Author's Astrocytic adenosine A2A receptors control the amyloid-β peptide-induced decrease of glutamate uptake‏
670 ‎‡a Author's Deletion of adenosine A2A receptors from astrocytes disrupts glutamate homeostasis leading to psychomotor and cognitive impairment: relevance to schizophrenia‏
670 ‎‡a Author's Ecto-5'-nucleotidase (CD73)-mediated formation of adenosine is critical for the striatal adenosine A2A receptor functions‏
670 ‎‡a Author's Modulation of astrocyte reactivity improves functional deficits in mouse models of Alzheimer's disease‏
670 ‎‡a Author's Oncostatin M promotes excitotoxicity by inhibiting glutamate uptake in astrocytes: implications in HIV-associated neurotoxicity‏
909 ‎‡a (orcid) 0000000151092062‏ ‎‡9 1‏
919 ‎‡a ecto5nucleotidasecd73mediatedformationofadenosineiscriticalforthestriataladenosinea2areceptorfunctions‏ ‎‡A Ecto-5'-nucleotidase (CD73)-mediated formation of adenosine is critical for the striatal adenosine A2A receptor functions‏ ‎‡9 1‏
919 ‎‡a astrocyticadenosinea2areceptorscontroltheamyloidβpeptideinduceddecreaseofglutamateuptake‏ ‎‡A Astrocytic adenosine A2A receptors control the amyloid-β peptide-induced decrease of glutamate uptake‏ ‎‡9 1‏
919 ‎‡a astrocytesdetectandupregulatetransmissionatinhibitorysynapsesofsomatostatininterneuronsontopyramidalcells‏ ‎‡A Astrocytes detect and upregulate transmission at inhibitory synapses of somatostatin interneurons onto pyramidal cells‏ ‎‡9 1‏
919 ‎‡a antagonisticinteractionbetweenadenosinea2areceptorsandna+k+atpaseα2controllingglutamateuptakeinastrocytes‏ ‎‡A Antagonistic interaction between adenosine A2A receptors and Na+/K+-ATPase-α2 controlling glutamate uptake in astrocytes‏ ‎‡9 1‏
919 ‎‡a amyloidbetapeptidedecreasesglutamateuptakeinculturedastrocytesinvolvementofoxidativestressandmitogenactivatedproteinkinasecascades‏ ‎‡A Amyloid-beta peptide decreases glutamate uptake in cultured astrocytes: involvement of oxidative stress and mitogen-activated protein kinase cascades‏ ‎‡9 1‏
919 ‎‡a adenosinea2areceptorsmodulateglutamateuptakeinculturedastrocytesandgliosomes‏ ‎‡A Adenosine A2A receptors modulate glutamate uptake in cultured astrocytes and gliosomes.‏ ‎‡9 1‏
919 ‎‡a deletionofadenosinea2areceptorsfromastrocytesdisruptsglutamatehomeostasisleadingtopsychomotorandcognitiveimpairmentrelevancetoschizophrenia‏ ‎‡A Deletion of adenosine A2A receptors from astrocytes disrupts glutamate homeostasis leading to psychomotor and cognitive impairment: relevance to schizophrenia‏ ‎‡9 1‏
919 ‎‡a oncostatin1000promotesexcitotoxicitybyinhibitingglutamateuptakeinastrocytesimplicationsinhivassociatedneurotoxicity‏ ‎‡A Oncostatin M promotes excitotoxicity by inhibiting glutamate uptake in astrocytes: implications in HIV-associated neurotoxicity‏ ‎‡9 1‏
919 ‎‡a modulationofastrocytereactivityimprovesfunctionaldeficitsinmousemodelsofalzheimersdisease‏ ‎‡A Modulation of astrocyte reactivity improves functional deficits in mouse models of Alzheimer's disease‏ ‎‡9 1‏
946 ‎‡a b‏ ‎‡9 1‏
996 ‎‡2 BLBNB|001587135
996 ‎‡2 LC|n 2023253565
996 ‎‡2 PLWABN|9810604862805606
996 ‎‡2 LC|n 2012213522
996 ‎‡2 BLBNB|000470719
996 ‎‡2 ISNI|0000000069519661
996 ‎‡2 LC|no2009171893
996 ‎‡2 BLBNB|000291459
996 ‎‡2 DNB|1236582772
996 ‎‡2 PTBNP|328686
996 ‎‡2 BLBNB|000318940
996 ‎‡2 ISNI|0000000070337283
996 ‎‡2 ISNI|0000000073205059
996 ‎‡2 PTBNP|946803
996 ‎‡2 DE633|pe30101057
996 ‎‡2 NTA|073331074
996 ‎‡2 ISNI|0000000071543142
996 ‎‡2 PTBNP|29645
996 ‎‡2 BLBNB|000236352
996 ‎‡2 LC|n 2019250712
996 ‎‡2 PTBNP|266033
996 ‎‡2 ISNI|0000000070329275
996 ‎‡2 BLBNB|001071856
996 ‎‡2 BIBSYS|5073418
996 ‎‡2 BLBNB|001494208
996 ‎‡2 DNB|1259635740
996 ‎‡2 ISNI|0000000109307066
996 ‎‡2 ISNI|0000000069119588
996 ‎‡2 DNB|1072865351
996 ‎‡2 LC|nb2009005106
996 ‎‡2 NTA|418887454
996 ‎‡2 NUKAT|n 2017033105
996 ‎‡2 SUDOC|070959900
996 ‎‡2 DNB|1244261904
996 ‎‡2 ISNI|0000000055600709
996 ‎‡2 LC|no2003028140
996 ‎‡2 PTBNP|788485
996 ‎‡2 PTBNP|1197051
996 ‎‡2 ISNI|0000000040326268
996 ‎‡2 LC|n 2017251050
996 ‎‡2 BNF|12906486
996 ‎‡2 BLBNB|001439208
996 ‎‡2 BLBNB|001458879
996 ‎‡2 BLBNB|000545867
996 ‎‡2 PTBNP|1393969
996 ‎‡2 NUKAT|n 2011062369
996 ‎‡2 LC|n 2005204258
996 ‎‡2 ISNI|0000000048127116
996 ‎‡2 DNB|127152830
996 ‎‡2 DNB|1207580767
996 ‎‡2 BLBNB|000291456
996 ‎‡2 PTBNP|152344
996 ‎‡2 PTBNP|1426933
996 ‎‡2 LC|no2010046936
997 ‎‡a 0 0 lived 0 0‏ ‎‡9 1‏