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WKP|Q59675103
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(Authority/Source Record)
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20241221010847.0 |
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(WKP)Q59675103
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0000-0003-2178-9218
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orcid
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(OCoLC)Q59675103
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Marcelo N N Vieira
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researcher ORCID ID = 0000-0003-2178-9218
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en
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iso5218
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Marcelo N N Vieira
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wetenschapper
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nl
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670
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Author's Amyloidogenicity and cytotoxicity of recombinant mature human islet amyloid polypeptide (rhIAPP).
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670
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Author's Connecting Alzheimer's disease to diabetes: Underlying mechanisms and potential therapeutic targets
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670
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Author's Formation of amyloid aggregates from human lysozyme and its disease-associated variants using hydrostatic pressure.
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670
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Author's Monoamine transporter inhibitors and norepinephrine reduce dopamine-dependent iron toxicity in cells derived from the substantia nigra
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670
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Author's Protection of synapses against Alzheimer's-linked toxins: insulin signaling prevents the pathogenic binding of Abeta oligomers
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670
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Author's Protein Tyrosine Phosphatase 1B (PTP1B): A Potential Target for Alzheimer's Therapy?
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670
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Author's Small molecule inhibitors of lysozyme amyloid aggregation
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670
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Author's Soluble oligomers from a non-disease related protein mimic Abeta-induced tau hyperphosphorylation and neurodegeneration
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670
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Author's Soluble protein oligomers as emerging toxins in Alzheimer's and other amyloid diseases.
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670
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Author's Training Microglia to Resist Alzheimer's Disease
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(orcid) 0000000321789218
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trainingmicrogliatoresistalzheimersdisease
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Training Microglia to Resist Alzheimer's Disease
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solubleproteinoligomersasemergingtoxinsinalzheimersandotheramyloiddiseases
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Soluble protein oligomers as emerging toxins in Alzheimer's and other amyloid diseases.
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solubleoligomersfromanondiseaserelatedproteinmimicabetainducedtauhyperphosphorylationandneurodegeneration
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Soluble oligomers from a non-disease related protein mimic Abeta-induced tau hyperphosphorylation and neurodegeneration
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1
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919
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smallmoleculeinhibitorsoflysozymeamyloidaggregation
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Small molecule inhibitors of lysozyme amyloid aggregation
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1
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proteintyrosinephosphatase1bptp1bapotentialtargetforalzheimerstherapy
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Protein Tyrosine Phosphatase 1B (PTP1B): A Potential Target for Alzheimer's Therapy?
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protectionofsynapsesagainstalzheimerslinkedtoxinsinsulinsignalingpreventsthepathogenicbindingofabetaoligomers
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Protection of synapses against Alzheimer's-linked toxins: insulin signaling prevents the pathogenic binding of Abeta oligomers
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919
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monoaminetransporterinhibitorsandnorepinephrinereducedopaminedependentirontoxicityincellsderivedfromthesubstantianigra
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Monoamine transporter inhibitors and norepinephrine reduce dopamine-dependent iron toxicity in cells derived from the substantia nigra
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1
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919
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formationofamyloidaggregatesfromhumanlysozymeanditsdiseaseassociatedvariantsusinghydrostaticpressure
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Formation of amyloid aggregates from human lysozyme and its disease-associated variants using hydrostatic pressure.
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1
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919
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connectingalzheimersdiseasetodiabetesunderlyingmechanismsandpotentialtherapeutictargets
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Connecting Alzheimer's disease to diabetes: Underlying mechanisms and potential therapeutic targets
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amyloidogenicityandcytotoxicityofrecombinantmaturehumanisletamyloidpolypeptiderhiapp
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Amyloidogenicity and cytotoxicity of recombinant mature human islet amyloid polypeptide (rhIAPP).
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