VIAF

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Leader 00000nz a2200037n 45 0
001 WKP|Q74261888 (VIAF cluster) (Authority/Source Record)
003 WKP
005 20241121000334.0
008 241121nneanz||abbn n and d
035 ‎‡a (WKP)Q74261888‏
024 ‎‡a 0000-0002-9473-0383‏ ‎‡2 orcid‏
035 ‎‡a (OCoLC)Q74261888‏
100 0 ‎‡a J L Hart‏ ‎‡c researcher‏ ‎‡9 en‏
400 0 ‎‡a J L Hart‏ ‎‡c wetenschapper‏ ‎‡9 nl‏
670 ‎‡a Author's 3',4'-Dihydroxyflavonol reduces superoxide and improves nitric oxide function in diabetic rat mesenteric arteries.‏
670 ‎‡a Author's 3',4'-Dihydroxyflavonol restores endothelium-dependent relaxation in small mesenteric artery from rats with type 1 and type 2 diabetes‏
670 ‎‡a Author's An evaluation of pharmacology curricula in Australian science and health-related degree programs‏
670 ‎‡a Author's An investigation of the mechanisms of hydrogen sulfide-induced vasorelaxation in rat middle cerebral arteries‏
670 ‎‡a Author's Chronic NaHS treatment decreases oxidative stress and improves endothelial function in diabetic mice‏
670 ‎‡a Author's Chronic NaHS Treatment Is Vasoprotective in High-Fat-Fed ApoE(-/-) Mice.‏
670 ‎‡a Author's Decreased vascular H2S production is associated with vascular oxidative stress in rats fed a high-fat western diet.‏
670 ‎‡a Author's Endothelium-dependent nitroxyl-mediated relaxation is resistant to superoxide anion scavenging and preserved in diabetic rat aorta‏
670 ‎‡a Author's H2S causes contraction and relaxation of major arteries of the rabbit.‏
670 ‎‡a Author's Hydrogen Sulfide in the RVLM and PVN has No Effect on Cardiovascular Regulation‏
670 ‎‡a Author's Hydrogen sulfide protects endothelial nitric oxide function under conditions of acute oxidative stress in vitro.‏
670 ‎‡a Author's Hydrogen sulfide treatment reduces blood pressure and oxidative stress in angiotensin II-induced hypertensive mice‏
670 ‎‡a Author's Influence of type-4 dipeptidyl peptidase inhibition on endothelium-dependent relaxation of aortae from a db/db mouse model of type 2 diabetes: a comparison with the effect of glimepiride‏
670 ‎‡a Author's Mechanism of vasorelaxation and role of endogenous hydrogen sulfide production in mouse aorta.‏
670 ‎‡a Author's Prevention of ischaemia-induced coronary vascular dysfunction.‏
670 ‎‡a Author's Vasorelaxation elicited by endogenous and exogenous hydrogen sulfide in mouse mesenteric arteries‏
909 ‎‡a (orcid) 0000000294730383‏ ‎‡9 1‏
919 ‎‡a 34dihydroxyflavonolreducessuperoxideandimprovesnitricoxidefunctionindiabeticratmesentericarteries‏ ‎‡A 3',4'-Dihydroxyflavonol reduces superoxide and improves nitric oxide function in diabetic rat mesenteric arteries.‏ ‎‡9 1‏
919 ‎‡a vasorelaxationelicitedbyendogenousandexogenoushydrogensulfideinmousemesentericarteries‏ ‎‡A Vasorelaxation elicited by endogenous and exogenous hydrogen sulfide in mouse mesenteric arteries‏ ‎‡9 1‏
919 ‎‡a preventionofischaemiainducedcoronaryvasculardysfunction‏ ‎‡A Prevention of ischaemia-induced coronary vascular dysfunction.‏ ‎‡9 1‏
919 ‎‡a mechanismofvasorelaxationandroleofendogenoushydrogensulfideproductioninmouseaorta‏ ‎‡A Mechanism of vasorelaxation and role of endogenous hydrogen sulfide production in mouse aorta.‏ ‎‡9 1‏
919 ‎‡a influenceoftype4dipeptidylpeptidaseinhibitiononendotheliumdependentrelaxationofaortaefromadbdbmousemodeloftype2diabetesacomparisonwiththeeffectofglimepiride‏ ‎‡A Influence of type-4 dipeptidyl peptidase inhibition on endothelium-dependent relaxation of aortae from a db/db mouse model of type 2 diabetes: a comparison with the effect of glimepiride‏ ‎‡9 1‏
919 ‎‡a hydrogensulfidetreatmentreducesbloodpressureandoxidativestressinangiotensin2inducedhypertensivemice‏ ‎‡A Hydrogen sulfide treatment reduces blood pressure and oxidative stress in angiotensin II-induced hypertensive mice‏ ‎‡9 1‏
919 ‎‡a hydrogensulfideprotectsendothelialnitricoxidefunctionunderconditionsofacuteoxidativestressinvitro‏ ‎‡A Hydrogen sulfide protects endothelial nitric oxide function under conditions of acute oxidative stress in vitro.‏ ‎‡9 1‏
919 ‎‡a hydrogensulfideinthervlmandpvnhasnoeffectoncardiovascularregulation‏ ‎‡A Hydrogen Sulfide in the RVLM and PVN has No Effect on Cardiovascular Regulation‏ ‎‡9 1‏
919 ‎‡a h2scausescontractionandrelaxationofmajorarteriesoftherabbit‏ ‎‡A H2S causes contraction and relaxation of major arteries of the rabbit.‏ ‎‡9 1‏
919 ‎‡a endotheliumdependentnitroxylmediatedrelaxationisresistanttosuperoxideanionscavengingandpreservedindiabeticrataorta‏ ‎‡A Endothelium-dependent nitroxyl-mediated relaxation is resistant to superoxide anion scavenging and preserved in diabetic rat aorta‏ ‎‡9 1‏
919 ‎‡a decreasedvascularh2sproductionisassociatedwithvascularoxidativestressinratsfedahighfatwesterndiet‏ ‎‡A Decreased vascular H2S production is associated with vascular oxidative stress in rats fed a high-fat western diet.‏ ‎‡9 1‏
919 ‎‡a chronicnahstreatmentisvasoprotectiveinhighfatfedapoemice‏ ‎‡A Chronic NaHS Treatment Is Vasoprotective in High-Fat-Fed ApoE(-/-) Mice.‏ ‎‡9 1‏
919 ‎‡a chronicnahstreatmentdecreasesoxidativestressandimprovesendothelialfunctionindiabeticmice‏ ‎‡A Chronic NaHS treatment decreases oxidative stress and improves endothelial function in diabetic mice‏ ‎‡9 1‏
919 ‎‡a investigationofthemechanismsofhydrogensulfideinducedvasorelaxationinratmiddlecerebralarteries‏ ‎‡A An investigation of the mechanisms of hydrogen sulfide-induced vasorelaxation in rat middle cerebral arteries‏ ‎‡9 1‏
919 ‎‡a evaluationofpharmacologycurriculainaustralianscienceandhealthrelateddegreeprograms‏ ‎‡A An evaluation of pharmacology curricula in Australian science and health-related degree programs‏ ‎‡9 1‏
919 ‎‡a 34dihydroxyflavonolrestoresendotheliumdependentrelaxationinsmallmesentericarteryfromratswithtype1andtype2diabetes‏ ‎‡A 3',4'-Dihydroxyflavonol restores endothelium-dependent relaxation in small mesenteric artery from rats with type 1 and type 2 diabetes‏ ‎‡9 1‏
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