VIAF

Virtual International Authority File

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Leader 00000nz a2200037n 45 0
001 WKP|Q88584784 (VIAF cluster) (Authority/Source Record)
003 WKP
005 20241020233208.0
008 241020nneanz||abbn n and d
035 ‎‡a (WKP)Q88584784‏
024 ‎‡a 0000-0001-6123-0481‏ ‎‡2 orcid‏
035 ‎‡a (OCoLC)Q88584784‏
100 0 ‎‡a Ana Carolina Proença da Fonseca‏ ‎‡c researcher‏ ‎‡9 en‏
400 0 ‎‡a Ana Carolina Proença da Fonseca‏ ‎‡c wetenschapper‏ ‎‡9 nl‏
670 ‎‡a Author's Adiponectin, Retinoic Acid Receptor Responder 2, and Peroxisome Proliferator-Activated Receptor-γ Coativator-1 Genes and the Risk for Obesity‏
670 ‎‡a Author's Genetic variants in the fat mass and obesity-associated (FTO) gene confer risk for extreme obesity and modulate adiposity in a Brazilian population‏
670 ‎‡a Author's Genetics of non-syndromic childhood obesity and the use of high-throughput DNA sequencing technologies.‏
670 ‎‡a Author's Identification of the First PAX4-MODY Family Reported in Brazil‏
670 ‎‡a Author's Severe phenotype in an apparent homozygosity caused by a large deletion in the CFTR gene: a case report‏
670 ‎‡a Author's Study of LEP, MRAP2 and POMC genes as potential causes of severe obesity in Brazilian patients‏
909 ‎‡a (orcid) 0000000161230481‏ ‎‡9 1‏
919 ‎‡a identificationofthe1pax4modyfamilyreportedinbrazil‏ ‎‡A Identification of the First PAX4-MODY Family Reported in Brazil‏ ‎‡9 1‏
919 ‎‡a geneticvariantsinthefatmassandobesityassociatedftogeneconferriskforextremeobesityandmodulateadiposityinabrazilianpopulation‏ ‎‡A Genetic variants in the fat mass and obesity-associated (FTO) gene confer risk for extreme obesity and modulate adiposity in a Brazilian population‏ ‎‡9 1‏
919 ‎‡a adiponectinretinoicacidreceptorresponder2andperoxisomeproliferatoractivatedreceptorγcoativator1genesandtheriskforobesity‏ ‎‡A Adiponectin, Retinoic Acid Receptor Responder 2, and Peroxisome Proliferator-Activated Receptor-γ Coativator-1 Genes and the Risk for Obesity‏ ‎‡9 1‏
919 ‎‡a geneticsofnonsyndromicchildhoodobesityandtheuseofhighthroughputdnasequencingtechnologies‏ ‎‡A Genetics of non-syndromic childhood obesity and the use of high-throughput DNA sequencing technologies.‏ ‎‡9 1‏
919 ‎‡a studyoflepmrap2andpomcgenesaspotentialcausesofsevereobesityinbrazilianpatients‏ ‎‡A Study of LEP, MRAP2 and POMC genes as potential causes of severe obesity in Brazilian patients‏ ‎‡9 1‏
919 ‎‡a severephenotypeinanapparenthomozygositycausedbyalargedeletioninthecftrgeneacasereport‏ ‎‡A Severe phenotype in an apparent homozygosity caused by a large deletion in the CFTR gene: a case report‏ ‎‡9 1‏
996 ‎‡2 NKC|nlk20020125673
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996 ‎‡2 LC|n 2019033038
996 ‎‡2 LC|n 2022251361
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996 ‎‡2 BIBSYS|90851195
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996 ‎‡2 LC|n 89183523
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996 ‎‡2 NTA|074786458
996 ‎‡2 RERO|A026962667
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996 ‎‡2 NTA|086762559
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996 ‎‡2 LIH|LNB:CYY3;=BU
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996 ‎‡2 BNF|15918785
996 ‎‡2 BNF|15784393
996 ‎‡2 DNB|1057700967
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996 ‎‡2 PTBNP|99947
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996 ‎‡2 LC|n 2023250794
996 ‎‡2 NUKAT|n 2009126455
996 ‎‡2 PTBNP|39113
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996 ‎‡2 CAOONL|ncf11475742
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996 ‎‡2 SELIBR|181899
996 ‎‡2 LC|n 86048620
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996 ‎‡2 CAOONL|ncf10690013
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996 ‎‡2 LC|nb2007008314
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996 ‎‡2 BNC|981058613207006706
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996 ‎‡2 BNCHL|10000000000000000259666
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996 ‎‡2 LIH|LNB:CO_a__q_;=B0
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996 ‎‡2 PLWABN|9811260772905606
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996 ‎‡2 CAOONL|ncf10550818
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996 ‎‡2 NYNYRILM|371469
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996 ‎‡2 LC|n 2006219038
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997 ‎‡a 0 0 lived 0 0‏ ‎‡9 1‏