Search
Leader | 00000nz a2200037n 45 0 | ||
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001 | WKP|Q90557231 (VIAF cluster) (Authority/Source Record) | ||
003 | WKP | ||
005 | 20241121000059.0 | ||
008 | 241121nneanz||abbn n and d | ||
035 | ‡a (WKP)Q90557231 | ||
024 | ‡a 0000-0002-7073-5067 ‡2 orcid | ||
024 | ‡a 57195244634 ‡2 scopus | ||
035 | ‡a (OCoLC)Q90557231 | ||
100 | 0 | ‡a Jose Luis Castañeda-Cabral ‡c researcher (ORCID 0000-0002-7073-5067) ‡9 en | |
400 | 0 | ‡a Jose Luis Castañeda-Cabral ‡c onderzoeker ‡9 nl | |
670 | ‡a Author's Glutamate Neonatal Excitotoxicity Modifies VEGF-A, VEGF-B, VEGFR-1 and VEGFR-2 Protein Expression Profiles During Postnatal Development of the Cerebral Cortex and Hippocampus of Male Rats | ||
670 | ‡a Author's Increased expression of proinflammatory cytokines and iNOS in the neocortical microvasculature of patients with temporal lobe epilepsy | ||
670 | ‡a Author's Increased protein expression of VEGF-A, VEGF-B, VEGF-C and their receptors in the temporal neocortex of pharmacoresistant temporal lobe epilepsy patients | ||
670 | ‡a Author's Neonatal excitotoxicity modifies blood-brain barrier properties increasing its susceptibility to hypertonic shock in adulthood | ||
670 | ‡a Author's Neuroprotective and Neurestorative Effects of Epo and VEGF: Perspectives for New Therapeutic Approaches to Neurological Diseases | ||
909 | ‡a (orcid) 0000000270735067 ‡9 1 | ||
909 | ‡a (scopus) 57195244634 ‡9 1 | ||
919 | ‡a increasedexpressionofproinflammatorycytokinesandinosintheneocorticalmicrovasculatureofpatientswithtemporallobeepilepsy ‡A Increased expression of proinflammatory cytokines and iNOS in the neocortical microvasculature of patients with temporal lobe epilepsy ‡9 1 | ||
919 | ‡a neuroprotectiveandneurestorativeeffectsofepoandvegfperspectivesfornewtherapeuticapproachestoneurologicaldiseases ‡A Neuroprotective and Neurestorative Effects of Epo and VEGF: Perspectives for New Therapeutic Approaches to Neurological Diseases ‡9 1 | ||
919 | ‡a neonatalexcitotoxicitymodifiesbloodbrainbarrierpropertiesincreasingitssusceptibilitytohypertonicshockinadulthood ‡A Neonatal excitotoxicity modifies blood-brain barrier properties increasing its susceptibility to hypertonic shock in adulthood ‡9 1 | ||
919 | ‡a increasedproteinexpressionofvegfavegfbvegf100andtheirreceptorsinthetemporalneocortexofpharmacoresistanttemporallobeepilepsypatients ‡A Increased protein expression of VEGF-A, VEGF-B, VEGF-C and their receptors in the temporal neocortex of pharmacoresistant temporal lobe epilepsy patients ‡9 1 | ||
919 | ‡a glutamateneonatalexcitotoxicitymodifiesvegfavegfbvegfr1andvegfr2proteinexpressionprofilesduringpostnataldevelopmentofthecerebralcortexandhippocampusofmalerats ‡A Glutamate Neonatal Excitotoxicity Modifies VEGF-A, VEGF-B, VEGFR-1 and VEGFR-2 Protein Expression Profiles During Postnatal Development of the Cerebral Cortex and Hippocampus of Male Rats ‡9 1 | ||
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