VIAF

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Leader 00000nz a2200037n 45 0
001 WKP|Q91264669 (VIAF cluster) (Authority/Source Record)
003 WKP
005 20241121000348.0
008 241121nneanz||abbn n and d
035 ‎‡a (WKP)Q91264669‏
024 ‎‡a 0000-0001-6491-2410‏ ‎‡2 orcid‏
035 ‎‡a (OCoLC)Q91264669‏
100 0 ‎‡a Ping-Hsien Lee‏ ‎‡c researcher (ORCID 0000-0001-6491-2410)‏ ‎‡9 en‏
400 0 ‎‡a Ping-Hsien Lee‏ ‎‡c wetenschapper‏ ‎‡9 nl‏
670 ‎‡a Author's A cleavage product of Polycystin-1 is a mitochondrial matrix protein that affects mitochondria morphology and function when heterologously expressed‏
670 ‎‡a Author's Host conditioning with IL-1β improves the antitumor function of adoptively transferred T cells‏
670 ‎‡a Author's Memory-phenotype CD4+ T cells spontaneously generated under steady-state conditions exert innate TH1-like effector function‏
670 ‎‡a Author's Mg2+ regulation of kinase signaling and immune function‏
670 ‎‡a Author's SARS-CoV-2 specific T-cells Are Rapidly Expanded for Therapeutic Use and Target Conserved Regions of Membrane Protein‏
670 ‎‡a Author's T cell stemness and dysfunction in tumors are triggered by a common mechanism‏
670 ‎‡a Author's T cells genetically engineered to overcome death signaling enhance adoptive cancer immunotherapy‏
909 ‎‡a (orcid) 0000000164912410‏ ‎‡9 1‏
919 ‎‡a hostconditioningwithil1βimprovestheantitumorfunctionofadoptivelytransferredtcells‏ ‎‡A Host conditioning with IL-1β improves the antitumor function of adoptively transferred T cells‏ ‎‡9 1‏
919 ‎‡a cleavageproductofpolycystin1isamitochondrialmatrixproteinthataffectsmitochondriamorphologyandfunctionwhenheterologouslyexpressed‏ ‎‡A A cleavage product of Polycystin-1 is a mitochondrial matrix protein that affects mitochondria morphology and function when heterologously expressed‏ ‎‡9 1‏
919 ‎‡a tcellsgeneticallyengineeredtoovercomedeathsignalingenhanceadoptivecancerimmunotherapy‏ ‎‡A T cells genetically engineered to overcome death signaling enhance adoptive cancer immunotherapy‏ ‎‡9 1‏
919 ‎‡a tcellstemnessanddysfunctionintumorsaretriggeredbyacommonmechanism‏ ‎‡A T cell stemness and dysfunction in tumors are triggered by a common mechanism‏ ‎‡9 1‏
919 ‎‡a sarscov2specifictcellsarerapidlyexpandedfortherapeuticuseandtargetconservedregionsofmembraneprotein‏ ‎‡A SARS-CoV-2 specific T-cells Are Rapidly Expanded for Therapeutic Use and Target Conserved Regions of Membrane Protein‏ ‎‡9 1‏
919 ‎‡a mg2+regulationofkinasesignalingandimmunefunction‏ ‎‡A Mg2+ regulation of kinase signaling and immune function‏ ‎‡9 1‏
919 ‎‡a memoryphenotypecd4+tcellsspontaneouslygeneratedundersteadystateconditionsexertinnateth1likeeffectorfunction‏ ‎‡A Memory-phenotype CD4+ T cells spontaneously generated under steady-state conditions exert innate TH1-like effector function‏ ‎‡9 1‏
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