VIAF

Virtual International Authority File

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Leader     00000nz a2200037n 45 0
001     WKP|Q42409760  (VIAF cluster)  (Authority/Source Record)
003     WKP
005     20241221010713.0
008     241221nneanz||abbn n and d
035 ‎‡a  (WKP)Q42409760‏
024 ‎‡a  0000-0002-3484-598X‏ ‎‡2  orcid‏
035 ‎‡a  (OCoLC)Q42409760‏
100 0 ‎‡a  Anna Pujol‏ ‎‡c  researcher‏ ‎‡9  en‏
375 ‎‡a  2‏ ‎‡2  iso5218‏
400 0 ‎‡a  Anna Pujol‏ ‎‡c  onderzoeker‏ ‎‡9  nl‏
400 0 ‎‡a  Anna Pujol‏ ‎‡c  investigadora‏ ‎‡9  es‏
400 0 ‎‡a  Anna Pujol‏ ‎‡c  investigadora‏ ‎‡9  ast‏
670 ‎‡a  Author's Adipose tissue overexpression of vascular endothelial growth factor protects against diet-induced obesity and insulin resistance.‏
670 ‎‡a  Author's Assessment of a dual regulatory role for NO in liver regeneration after partial hepatectomy: protection against apoptosis and retardation of hepatocyte proliferation‏
670 ‎‡a  Author's Chronically increased glucose uptake by adipose tissue leads to lactate production and improved insulin sensitivity rather than obesity in the mouse‏
670 ‎‡a  Author's Correction of diabetic alterations by glucokinase.‏
670 ‎‡a  Author's Counteraction of type 1 diabetic alterations by engineering skeletal muscle to produce insulin: insights from transgenic mice.‏
670 ‎‡a  Author's Disease correction by AAV-mediated gene therapy in a new mouse model of mucopolysaccharidosis type IIID.‏
670 ‎‡a  Author's Enforced expression of protein kinase C in skeletal muscle causes physical inactivity, fatty liver and insulin resistance in the brain‏
670 ‎‡a  Author's Evidence from Transgenic Mice That Interferon-β May Be Involved in the Onset of Diabetes Mellitus‏
670 ‎‡a  Author's Evidence from transgenic mice that myc regulates hepatic glycolysis‏
670 ‎‡a  Author's Expression of Glucokinase in Skeletal Muscle: A New Approach to Counteract Diabetic Hyperglycemia‏
670 ‎‡a  Author's Expression of GLUT-2 antisense RNA in beta cells of transgenic mice leads to diabetes‏
670 ‎‡a  Author's HMGA1 overexpression in adipose tissue impairs adipogenesis and prevents diet-induced obesity and insulin resistance.‏
670 ‎‡a  Author's Increased fatty acid re-esterification by PEPCK overexpression in adipose tissue leads to obesity without insulin resistance‏
670 ‎‡a  Author's Overexpression of mitochondrial 3-hydroxy-3-methylglutaryl-CoA synthase in transgenic mice causes hepatic hyperketogenesis‏
670 ‎‡a  Author's Overexpression of ubiquitous 6-phosphofructo-2-kinase in the liver of transgenic mice results in weight gain.‏
670 ‎‡a  Author's Phosphofructo-1-kinase deficiency leads to a severe cardiac and hematological disorder in addition to skeletal muscle glycogenosis‏
670 ‎‡a  Author's Progressive neurologic and somatic disease in a novel mouse model of human mucopolysaccharidosis type IIIC.‏
670 ‎‡a  Author's Protein kinase Cδ regulates nuclear export of FOXO1 through phosphorylation of the chaperone 14-3-3ζ.‏
670 ‎‡a  Author's Regulated expression of human insulin in the liver of transgenic mice corrects diabetic alterations.‏
670 ‎‡a  Author's The suppressor of cytokine signalling 2‏
670 ‎‡a  Author's The suppressor of cytokine signalling 2 (SOCS2) is a key repressor of insulin secretion.‏
670 ‎‡a  Author's TRANSGENIC MICE IN THE ANALYSIS OF METABOLIC REGULATION‏
670 ‎‡a  Author's Transgenic mice overexpressing human G972R IRS-1 show impaired insulin action and insulin secretion.‏
670 ‎‡a  Author's Transgenic mice overexpressing insulin-like growth factor-II in beta cells develop type 2 diabetes.‏
670 ‎‡a  Author's Transgenic mice overexpressing phosphoenolpyruvate carboxykinase develop non-insulin-dependent diabetes mellitus.‏
670 ‎‡a  Author's Vascular endothelial growth factor-mediated islet hypervascularization and inflammation contribute to progressive reduction of β-cell mass.‏
670 ‎‡a  Author's Wnt pathway modulation generates blastomere-derived mouse embryonic stem cells with different pluripotency features‏
909 ‎‡a  (orcid) 000000023484598x‏ ‎‡9  1‏
919 ‎‡a  enforcedexpressionofproteinkinase100inskeletalmusclecausesphysicalinactivityfattyliverandinsulinresistanceinthebrain‏ ‎‡A  Enforced expression of protein kinase C in skeletal muscle causes physical inactivity, fatty liver and insulin resistance in the brain‏ ‎‡9  1‏
919 ‎‡a  evidencefromtransgenicmicethatinterferonβmaybeinvolvedintheonsetofdiabetesmellitus‏ ‎‡A  Evidence from Transgenic Mice That Interferon-β May Be Involved in the Onset of Diabetes Mellitus‏ ‎‡9  1‏
919 ‎‡a  evidencefromtransgenicmicethatmycregulateshepaticglycolysis‏ ‎‡A  Evidence from transgenic mice that myc regulates hepatic glycolysis‏ ‎‡9  1‏
919 ‎‡a  assessmentofadualregulatoryrolefornoinliverregenerationafterpartialhepatectomyprotectionagainstapoptosisandretardationofhepatocyteproliferation‏ ‎‡A  Assessment of a dual regulatory role for NO in liver regeneration after partial hepatectomy: protection against apoptosis and retardation of hepatocyte proliferation‏ ‎‡9  1‏
919 ‎‡a  adiposetissueoverexpressionofvascularendothelialgrowthfactorprotectsagainstdietinducedobesityandinsulinresistance‏ ‎‡A  Adipose tissue overexpression of vascular endothelial growth factor protects against diet-induced obesity and insulin resistance.‏ ‎‡9  1‏
919 ‎‡a  expressionofglucokinaseinskeletalmuscleanewapproachtocounteractdiabetichyperglycemia‏ ‎‡A  Expression of Glucokinase in Skeletal Muscle: A New Approach to Counteract Diabetic Hyperglycemia‏ ‎‡9  1‏
919 ‎‡a  expressionofglut2antisensernainbetacellsoftransgenicmiceleadstodiabetes‏ ‎‡A  Expression of GLUT-2 antisense RNA in beta cells of transgenic mice leads to diabetes‏ ‎‡9  1‏
919 ‎‡a  hmga1overexpressioninadiposetissueimpairsadipogenesisandpreventsdietinducedobesityandinsulinresistance‏ ‎‡A  HMGA1 overexpression in adipose tissue impairs adipogenesis and prevents diet-induced obesity and insulin resistance.‏ ‎‡9  1‏
919 ‎‡a  increasedfattyacidreesterificationbypepckoverexpressioninadiposetissueleadstoobesitywithoutinsulinresistance‏ ‎‡A  Increased fatty acid re-esterification by PEPCK overexpression in adipose tissue leads to obesity without insulin resistance‏ ‎‡9  1‏
919 ‎‡a  overexpressionofmitochondrial3hydroxy3methylglutarylcoasynthaseintransgenicmicecauseshepatichyperketogenesis‏ ‎‡A  Overexpression of mitochondrial 3-hydroxy-3-methylglutaryl-CoA synthase in transgenic mice causes hepatic hyperketogenesis‏ ‎‡9  1‏
919 ‎‡a  overexpressionofubiquitous6phosphofructo2kinaseintheliveroftransgenicmiceresultsinweightgain‏ ‎‡A  Overexpression of ubiquitous 6-phosphofructo-2-kinase in the liver of transgenic mice results in weight gain.‏ ‎‡9  1‏
919 ‎‡a  phosphofructo1kinasedeficiencyleadstoaseverecardiacandhematologicaldisorderinadditiontoskeletalmuscleglycogenosis‏ ‎‡A  Phosphofructo-1-kinase deficiency leads to a severe cardiac and hematological disorder in addition to skeletal muscle glycogenosis‏ ‎‡9  1‏
919 ‎‡a  progressiveneurologicandsomaticdiseaseinanovelmousemodelofhumanmucopolysaccharidosistypeiiic‏ ‎‡A  Progressive neurologic and somatic disease in a novel mouse model of human mucopolysaccharidosis type IIIC.‏ ‎‡9  1‏
919 ‎‡a  proteinkinasecδregulatesnuclearexportoffoxo1throughphosphorylationofthechaperone1433ζ‏ ‎‡A  Protein kinase Cδ regulates nuclear export of FOXO1 through phosphorylation of the chaperone 14-3-3ζ.‏ ‎‡9  1‏
919 ‎‡a  regulatedexpressionofhumaninsulinintheliveroftransgenicmicecorrectsdiabeticalterations‏ ‎‡A  Regulated expression of human insulin in the liver of transgenic mice corrects diabetic alterations.‏ ‎‡9  1‏
919 ‎‡a  suppressorofcytokinesignalling2‏ ‎‡A  The suppressor of cytokine signalling 2‏ ‎‡9  1‏
919 ‎‡a  suppressorofcytokinesignalling2socs2isakeyrepressorofinsulinsecretion‏ ‎‡A  The suppressor of cytokine signalling 2 (SOCS2) is a key repressor of insulin secretion.‏ ‎‡9  1‏
919 ‎‡a  transgenicmiceintheanalysisofmetabolicregulation‏ ‎‡A  TRANSGENIC MICE IN THE ANALYSIS OF METABOLIC REGULATION‏ ‎‡9  1‏
919 ‎‡a  transgenicmiceoverexpressinghumang972rirs1showimpairedinsulinactionandinsulinsecretion‏ ‎‡A  Transgenic mice overexpressing human G972R IRS-1 show impaired insulin action and insulin secretion.‏ ‎‡9  1‏
919 ‎‡a  transgenicmiceoverexpressinginsulinlikegrowthfactor2inbetacellsdeveloptype2diabetes‏ ‎‡A  Transgenic mice overexpressing insulin-like growth factor-II in beta cells develop type 2 diabetes.‏ ‎‡9  1‏
919 ‎‡a  transgenicmiceoverexpressingphosphoenolpyruvatecarboxykinasedevelopnoninsulindependentdiabetesmellitus‏ ‎‡A  Transgenic mice overexpressing phosphoenolpyruvate carboxykinase develop non-insulin-dependent diabetes mellitus.‏ ‎‡9  1‏
919 ‎‡a  vascularendothelialgrowthfactormediatedislethypervascularizationandinflammationcontributetoprogressivereductionofβcellmass‏ ‎‡A  Vascular endothelial growth factor-mediated islet hypervascularization and inflammation contribute to progressive reduction of β-cell mass.‏ ‎‡9  1‏
919 ‎‡a  wntpathwaymodulationgeneratesblastomerederivedmouseembryonicstemcellswithdifferentpluripotencyfeatures‏ ‎‡A  Wnt pathway modulation generates blastomere-derived mouse embryonic stem cells with different pluripotency features‏ ‎‡9  1‏
919 ‎‡a  chronicallyincreasedglucoseuptakebyadiposetissueleadstolactateproductionandimprovedinsulinsensitivityratherthanobesityinthemouse‏ ‎‡A  Chronically increased glucose uptake by adipose tissue leads to lactate production and improved insulin sensitivity rather than obesity in the mouse‏ ‎‡9  1‏
919 ‎‡a  correctionofdiabeticalterationsbyglucokinase‏ ‎‡A  Correction of diabetic alterations by glucokinase.‏ ‎‡9  1‏
919 ‎‡a  counteractionoftype1diabeticalterationsbyengineeringskeletalmuscletoproduceinsulininsightsfromtransgenicmice‏ ‎‡A  Counteraction of type 1 diabetic alterations by engineering skeletal muscle to produce insulin: insights from transgenic mice.‏ ‎‡9  1‏
919 ‎‡a  diseasecorrectionbyaavmediatedgenetherapyinanewmousemodelofmucopolysaccharidosistypeiiid‏ ‎‡A  Disease correction by AAV-mediated gene therapy in a new mouse model of mucopolysaccharidosis type IIID.‏ ‎‡9  1‏
946 ‎‡a  a‏ ‎‡9  1‏
996 ‎‡2  BIBSYS|6002987‏
996 ‎‡2  DNB|1157296343‏
996 ‎‡2  ISNI|0000000497007670‏
996 ‎‡2  BNC|981058510999006706‏
996 ‎‡2  BLBNB|000536615‏
996 ‎‡2  BNC|981058515413006706‏
996 ‎‡2  PTBNP|1770788‏
996 ‎‡2  BNC|981058514618006706‏
996 ‎‡2  BNC|981058512518206706‏
996 ‎‡2  BNE|XX970123‏
996 ‎‡2  BNE|XX896197‏
996 ‎‡2  BNC|981061113072506706‏
996 ‎‡2  BNC|981058612182506706‏
996 ‎‡2  CAOONL|ncf10686827‏
996 ‎‡2  ISNI|0000000059538471‏
996 ‎‡2  BNCHL|10000000000000000270401‏
996 ‎‡2  ISNI|0000000509874801‏
996 ‎‡2  BNF|15373552‏
996 ‎‡2  RERO|A003716384‏
996 ‎‡2  BNC|981058608319906706‏
996 ‎‡2  ISNI|0000000060860986‏
996 ‎‡2  BNE|XX1710509‏
996 ‎‡2  BNC|981058518055406706‏
996 ‎‡2  NTA|070085021‏
996 ‎‡2  BNE|XX1058305‏
996 ‎‡2  BLBNB|000375065‏
996 ‎‡2  ISNI|0000000033148723‏
996 ‎‡2  BNC|981058617055606706‏
996 ‎‡2  ISNI|000000005944204X‏
996 ‎‡2  BNE|XX5596181‏
996 ‎‡2  LC|no2006046849‏
996 ‎‡2  BNC|981058595283206706‏
996 ‎‡2  ISNI|0000000477514958‏
996 ‎‡2  SUDOC|112337384‏
996 ‎‡2  RERO|A013907213‏
996 ‎‡2  SUDOC|264966961‏
996 ‎‡2  BNC|981058527500506706‏
996 ‎‡2  BNC|981058511002006706‏
996 ‎‡2  SUDOC|232718237‏
996 ‎‡2  RERO|A027772031‏
996 ‎‡2  SUDOC|260327921‏
996 ‎‡2  DNB|105770122X‏
996 ‎‡2  BNF|17782208‏
996 ‎‡2  SUDOC|128482001‏
996 ‎‡2  LC|n 91071799‏
996 ‎‡2  BNE|XX1557153‏
996 ‎‡2  SUDOC|134957628‏
996 ‎‡2  SUDOC|252932277‏
996 ‎‡2  PLWABN|9810551682405606‏
996 ‎‡2  BNE|XX1273140‏
996 ‎‡2  BNC|981058514121506706‏
996 ‎‡2  BNF|13592913‏
996 ‎‡2  BNF|12119162‏
996 ‎‡2  BNF|18063031‏
996 ‎‡2  LC|no 98067126‏
996 ‎‡2  BIBSYS|98029185‏
996 ‎‡2  SUDOC|250451530‏
996 ‎‡2  SUDOC|260562688‏
996 ‎‡2  RERO|A000144114‏
997 ‎‡a  0 0 lived 0 0‏ ‎‡9  1‏