|
Leader
|
|
|
00000nz a2200037n 45 0 |
|
001
|
|
|
WKP|Q58339500
(VIAF cluster)
(Authority/Source Record)
|
|
003
|
|
|
WKP |
|
005
|
|
|
20241221010722.0 |
|
008
|
|
|
241221nneanz||abbn n and d |
|
035
|
|
|
‡a
(WKP)Q58339500
|
|
024
|
|
|
‡a
0000-0002-3043-8797
‡2
orcid
|
|
024
|
|
|
‡a
6504669524
‡2
scopus
|
|
035
|
|
|
‡a
(OCoLC)Q58339500
|
|
100
|
0 |
|
‡a
Luis Federico Batiz
‡c
researcher, ORCID id # 0000-0002-3043-8797
‡9
en
|
|
375
|
|
|
‡a
1
‡2
iso5218
|
|
400
|
0 |
|
‡a
Luis Federico Batiz
‡c
wetenschapper
‡9
nl
|
|
670
|
|
|
‡a
Author's A cell junction pathology of neural stem cells leads to abnormal neurogenesis and hydrocephalus.
|
|
670
|
|
|
‡a
Author's A Role for mir-26a in Stress: A Potential sEV Biomarker and Modulator of Excitatory Neurotransmission
|
|
670
|
|
|
‡a
Author's A simple PCR-based genotyping method for M105I mutation of alpha-SNAP enhances the study of early pathological changes in hyh phenotype.
|
|
670
|
|
|
‡a
Author's Aging restricts the ability of mesenchymal stem cells to promote the generation of oligodendrocytes during remyelination
|
|
670
|
|
|
‡a
Author's Astrocytes at the Hub of the Stress Response: Potential Modulation of Neurogenesis by miRNAs in Astrocyte-Derived Exosomes.
|
|
670
|
|
|
‡a
Author's Cellular mechanisms involved in the stenosis and obliteration of the cerebral aqueduct of hyh mutant mice developing congenital hydrocephalus.
|
|
670
|
|
|
‡a
Author's Defects in cell-cell junctions lead to neuroepithelial/ependymal denudation in the telencephalon of human hydrocephalic foetuses
|
|
670
|
|
|
‡a
Author's Disruption of neural progenitors along the ventricular and subventricular zones in periventricular heterotopia
|
|
670
|
|
|
‡a
Author's Disruption of the neurogenic niche in the subventricular zone of postnatal hydrocephalic hyh mice.
|
|
670
|
|
|
‡a
Author's Heterogeneous expression of hydrocephalic phenotype in the hyh mice carrying a point mutation in alpha-SNAP.
|
|
670
|
|
|
‡a
Author's Molecular mechanisms underlying neuroepithelial/ependymal denudation in the hydrocephalic hyh mutant: spatial and temporal expression of alpha-SNAP and N-cadherin.
|
|
670
|
|
|
‡a
Author's Neuroependymal denudation is in progress in full-term human foetal spina bifida aperta
|
|
670
|
|
|
‡a
Author's New ependymal cells are born postnatally in two discrete regions of the mouse brain and support ventricular enlargement in hydrocephalus.
|
|
670
|
|
|
‡a
Author's Patterned neuropathologic events occurring in hyh congenital hydrocephalic mutant mice.
|
|
670
|
|
|
‡a
Author's Publisher Correction: α-SNAP is expressed in mouse ovarian granulosa cells and plays a key role in folliculogenesis and female fertility
|
|
670
|
|
|
‡a
Author's Role of adherens junctions and apical-basal polarity of neural stem/progenitor cells in the pathogenesis of neurodevelopmental disorders: a novel perspective on congenital Zika syndrome
|
|
670
|
|
|
‡a
Author's Sperm from hyh mice carrying a point mutation in alphaSNAP have a defect in acrosome reaction
|
|
670
|
|
|
‡a
Author's α-SNAP is expressed in mouse ovarian granulosa cells and plays a key role in folliculogenesis and female fertility
|
|
909
|
|
|
‡a
(scopus) 6504669524
‡9
1
|
|
909
|
|
|
‡a
(orcid) 0000000230438797
‡9
1
|
|
919
|
|
|
‡a
αsnapisexpressedinmouseovariangranulosacellsandplaysakeyroleinfolliculogenesisandfemalefertility
‡A
α-SNAP is expressed in mouse ovarian granulosa cells and plays a key role in folliculogenesis and female fertility
‡9
1
|
|
919
|
|
|
‡a
roleofadherensjunctionsandapicalbasalpolarityofneuralstemprogenitorcellsinthepathogenesisofneurodevelopmentaldisordersanovelperspectiveoncongenitalzikasyndrome
‡A
Role of adherens junctions and apical-basal polarity of neural stem/progenitor cells in the pathogenesis of neurodevelopmental disorders: a novel perspective on congenital Zika syndrome
‡9
1
|
|
919
|
|
|
‡a
publishercorrectionαsnapisexpressedinmouseovariangranulosacellsandplaysakeyroleinfolliculogenesisandfemalefertility
‡A
Publisher Correction: α-SNAP is expressed in mouse ovarian granulosa cells and plays a key role in folliculogenesis and female fertility
‡9
1
|
|
919
|
|
|
‡a
patternedneuropathologiceventsoccurringinhyhcongenitalhydrocephalicmutantmice
‡A
Patterned neuropathologic events occurring in hyh congenital hydrocephalic mutant mice.
‡9
1
|
|
919
|
|
|
‡a
newependymalcellsarebornpostnatallyin2discreteregionsofthemousebrainandsupportventricularenlargementinhydrocephalus
‡A
New ependymal cells are born postnatally in two discrete regions of the mouse brain and support ventricular enlargement in hydrocephalus.
‡9
1
|
|
919
|
|
|
‡a
neuroependymaldenudationisinprogressinfulltermhumanfoetalspinabifidaaperta
‡A
Neuroependymal denudation is in progress in full-term human foetal spina bifida aperta
‡9
1
|
|
919
|
|
|
‡a
molecularmechanismsunderlyingneuroepithelialependymaldenudationinthehydrocephalichyhmutantspatialandtemporalexpressionofalphasnapandncadherin
‡A
Molecular mechanisms underlying neuroepithelial/ependymal denudation in the hydrocephalic hyh mutant: spatial and temporal expression of alpha-SNAP and N-cadherin.
‡9
1
|
|
919
|
|
|
‡a
heterogeneousexpressionofhydrocephalicphenotypeinthehyhmicecarryingapointmutationinalphasnap
‡A
Heterogeneous expression of hydrocephalic phenotype in the hyh mice carrying a point mutation in alpha-SNAP.
‡9
1
|
|
919
|
|
|
‡a
disruptionoftheneurogenicnicheinthesubventricularzoneofpostnatalhydrocephalichyhmice
‡A
Disruption of the neurogenic niche in the subventricular zone of postnatal hydrocephalic hyh mice.
‡9
1
|
|
919
|
|
|
‡a
disruptionofneuralprogenitorsalongtheventricularandsubventricularzonesinperiventricularheterotopia
‡A
Disruption of neural progenitors along the ventricular and subventricular zones in periventricular heterotopia
‡9
1
|
|
919
|
|
|
‡a
defectsincellcelljunctionsleadtoneuroepithelialependymaldenudationinthetelencephalonofhumanhydrocephalicfoetuses
‡A
Defects in cell-cell junctions lead to neuroepithelial/ependymal denudation in the telencephalon of human hydrocephalic foetuses
‡9
1
|
|
919
|
|
|
‡a
cellularmechanismsinvolvedinthestenosisandobliterationofthecerebralaqueductofhyhmutantmicedevelopingcongenitalhydrocephalus
‡A
Cellular mechanisms involved in the stenosis and obliteration of the cerebral aqueduct of hyh mutant mice developing congenital hydrocephalus.
‡9
1
|
|
919
|
|
|
‡a
astrocytesatthehubofthestressresponsepotentialmodulationofneurogenesisbymirnasinastrocytederivedexosomes
‡A
Astrocytes at the Hub of the Stress Response: Potential Modulation of Neurogenesis by miRNAs in Astrocyte-Derived Exosomes.
‡9
1
|
|
919
|
|
|
‡a
agingrestrictstheabilityofmesenchymalstemcellstopromotethegenerationofoligodendrocytesduringremyelination
‡A
Aging restricts the ability of mesenchymal stem cells to promote the generation of oligodendrocytes during remyelination
‡9
1
|
|
919
|
|
|
‡a
simplepcrbasedgenotypingmethodform105imutationofalphasnapenhancesthestudyofearlypathologicalchangesinhyhphenotype
‡A
A simple PCR-based genotyping method for M105I mutation of alpha-SNAP enhances the study of early pathological changes in hyh phenotype.
‡9
1
|
|
919
|
|
|
‡a
roleformir26ainstressapotentialsevbiomarkerandmodulatorofexcitatoryneurotransmission
‡A
A Role for mir-26a in Stress: A Potential sEV Biomarker and Modulator of Excitatory Neurotransmission
‡9
1
|
|
919
|
|
|
‡a
celljunctionpathologyofneuralstemcellsleadstoabnormalneurogenesisandhydrocephalus
‡A
A cell junction pathology of neural stem cells leads to abnormal neurogenesis and hydrocephalus.
‡9
1
|
|
919
|
|
|
‡a
spermfromhyhmicecarryingapointmutationinalphasnaphaveadefectinacrosomereaction
‡A
Sperm from hyh mice carrying a point mutation in alphaSNAP have a defect in acrosome reaction
‡9
1
|
|
946
|
|
|
‡a
b
‡9
1
|
|
996
|
|
|
‡2
J9U|987007426668605171
|
|
996
|
|
|
‡2
ISNI|0000000116734285
|
|
996
|
|
|
‡2
LNB|LNC10-000156960
|
|
996
|
|
|
‡2
NII|DA04487053
|
|
996
|
|
|
‡2
BIBSYS|90127846
|
|
996
|
|
|
‡2
LC|n 84030769
|
|
996
|
|
|
‡2
DNB|131427407
|
|
996
|
|
|
‡2
NTA|071878947
|
|
996
|
|
|
‡2
RERO|A003749644
|
|
996
|
|
|
‡2
CAOONL|ncf11716351
|
|
996
|
|
|
‡2
SUDOC|078699835
|
|
996
|
|
|
‡2
NUKAT|n 01029962
|
|
996
|
|
|
‡2
BNCHL|10000000000000000271025
|
|
997
|
|
|
‡a
0 0 lived 0 0
‡9
1
|
