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WKP|Q80072663
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(WKP)Q80072663
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0000-0001-7574-2162
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orcid
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14824443200
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scopus
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(OCoLC)Q80072663
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100
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জেমস ই ক্লার্ক
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bn
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375
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1
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iso5218
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James E. Clark
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researcher
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en
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James E Clark
‡c
wetenschapper
‡9
nl
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670
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Author's A chemical genetic approach reveals that p38alpha MAPK activation by diphosphorylation aggravates myocardial infarction and is prevented by the direct binding of SB203580.
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670
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Author's A Novel α-Calcitonin Gene-Related Peptide Analogue Protects Against End-Organ Damage in Experimental Hypertension, Cardiac Hypertrophy, and Heart Failure.
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670
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Author's Advancements in pressure-volume catheter technology - stress remodelling after infarction.
|
670
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‡a
Author's Aged-senescent cells contribute to impaired heart regeneration.
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670
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‡a
Author's Author Correction: Non-Invasive whole-body detection of complement activation using radionuclide imaging in a mouse model of myocardial ischaemia-reperfusion injury.
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670
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‡a
Author's Cardiac hypertrophy in mice expressing unphosphorylatable phospholemman
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670
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‡a
Author's Cardiac myosin-binding protein C: a potential early biomarker of myocardial injury.
|
670
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‡a
Author's Cardioprotection by an anti-MASP-2 antibody in a murine model of myocardial infarction
|
670
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‡a
Author's Constitutive glycogen synthase kinase-3alpha/beta activity protects against chronic beta-adrenergic remodelling of the heart.
|
670
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‡a
Author's Detection of anthracycline-induced cardiotoxicity using perfusion-corrected Tc sestamibi SPECT
|
670
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‡a
Author's Epistatic rescue of Nkx2.5 adult cardiac conduction disease phenotypes by prospero-related homeobox protein 1 and HDAC3.
|
670
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‡a
Author's Experimental hyperleptinemia in neonatal rats leads to selective leptin responsiveness, hypertension, and altered myocardial function
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670
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‡a
Author's Glycogen synthase kinase-3 inactivation is not required for ischemic preconditioning or postconditioning in the mouse
|
670
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‡a
Author's Investigating the potential role of TRPA1 in locomotion and cardiovascular control during hypertension.
|
670
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‡a
Author's Loss of Protein Kinase Novel 1 (PKN1) is associated with mild systolic and diastolic contractile dysfunction, increased phospholamban Thr17 phosphorylation, and exacerbated ischaemia-reperfusion injury
|
670
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‡a
Author's Mechanism and consequence of the autoactivation of p38α mitogen-activated protein kinase promoted by TAB1
|
670
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‡a
Author's Moving in extreme environments: inert gas narcosis and underwater activities.
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670
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Author's Myocardial stress remodelling after regional infarction is independent of glycogen synthase kinase-3 inactivation.
|
670
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Author's Non-Invasive whole-body detection of complement activation using radionuclide imaging in a mouse model of myocardial ischaemia-reperfusion injury.
|
670
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Author's Overexpression of the transcription factor Hand1 causes predisposition towards arrhythmia in mice.
|
670
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‡a
Author's Patent foramen ovale and scuba diving: a practical guide for physicians on when to refer for screening
|
670
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Author's Post-infarction remodeling is independent of mitogen-activated protein kinase kinase 3
|
670
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Author's Post-infarction remodeling is independent of mitogen-activated protein kinase kinase 3 (MKK3)
|
670
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‡a
Author's Pravastatin therapy during preeclampsia prevents long-term adverse health effects in mice.
|
670
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‡a
Author's Response to role of hyperleptinemia in the regulation of blood pressure and cardiac function
|
670
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‡a
Author's Targeting of mannan-binding lectin-associated serine protease-2 confers protection from myocardial and gastrointestinal ischemia/reperfusion injury.
|
670
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‡a
Author's The POU4F2/Brn-3b transcription factor is required for the hypertrophic response to angiotensin II in the heart
|
670
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Author's The TAB1-p38α complex aggravates myocardial injury and can be targeted by small molecules
|
670
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|
‡a
Author's Thymosin β4 facilitates epicardial neovascularization of the injured adult heart
|
670
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‡a
Author's Tissue acidosis does not mediate the hypoxia selectivity of [Cu][Cu
|
670
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‡a
Author's Tissue acidosis does not mediate the hypoxia selectivity of [Cu][Cu(ATSM)] in the isolated perfused rat heart
|
670
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‡a
Author's Trapped platelets activated in ischemia initiate ventricular fibrillation
|
909
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‡a
(scopus) 14824443200
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1
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909
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‡a
(orcid) 0000000175742162
‡9
1
|
919
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‡a
glycogensynthasekinase3inactivationisnotrequiredforischemicpreconditioningorpostconditioninginthemouse
‡A
Glycogen synthase kinase-3 inactivation is not required for ischemic preconditioning or postconditioning in the mouse
‡9
1
|
919
|
|
|
‡a
experimentalhyperleptinemiainneonatalratsleadstoselectiveleptinresponsivenesshypertensionandalteredmyocardialfunction
‡A
Experimental hyperleptinemia in neonatal rats leads to selective leptin responsiveness, hypertension, and altered myocardial function
‡9
1
|
919
|
|
|
‡a
epistaticrescueofnkx25adultcardiacconductiondiseasephenotypesbyprosperorelatedhomeoboxprotein1andhdac3
‡A
Epistatic rescue of Nkx2.5 adult cardiac conduction disease phenotypes by prospero-related homeobox protein 1 and HDAC3.
‡9
1
|
919
|
|
|
‡a
detectionofanthracyclineinducedcardiotoxicityusingperfusioncorrectedtcsestamibispect
‡A
Detection of anthracycline-induced cardiotoxicity using perfusion-corrected Tc sestamibi SPECT
‡9
1
|
919
|
|
|
‡a
constitutiveglycogensynthasekinase3alphabetaactivityprotectsagainstchronicbetaadrenergicremodellingoftheheart
‡A
Constitutive glycogen synthase kinase-3alpha/beta activity protects against chronic beta-adrenergic remodelling of the heart.
‡9
1
|
919
|
|
|
‡a
cardioprotectionbyanantimasp2antibodyinamurinemodelofmyocardialinfarction
‡A
Cardioprotection by an anti-MASP-2 antibody in a murine model of myocardial infarction
‡9
1
|
919
|
|
|
‡a
cardiacmyosinbindingprotein100apotentialearlybiomarkerofmyocardialinjury
‡A
Cardiac myosin-binding protein C: a potential early biomarker of myocardial injury.
‡9
1
|
919
|
|
|
‡a
cardiachypertrophyinmiceexpressingunphosphorylatablephospholemman
‡A
Cardiac hypertrophy in mice expressing unphosphorylatable phospholemman
‡9
1
|
919
|
|
|
‡a
authorcorrectionnoninvasivewholebodydetectionofcomplementactivationusingradionuclideimaginginamousemodelofmyocardialischaemiareperfusioninjury
‡A
Author Correction: Non-Invasive whole-body detection of complement activation using radionuclide imaging in a mouse model of myocardial ischaemia-reperfusion injury.
‡9
1
|
919
|
|
|
‡a
agedsenescentcellscontributetoimpairedheartregeneration
‡A
Aged-senescent cells contribute to impaired heart regeneration.
‡9
1
|
919
|
|
|
‡a
advancementsinpressurevolumecathetertechnologystressremodellingafterinfarction
‡A
Advancements in pressure-volume catheter technology - stress remodelling after infarction.
‡9
1
|
919
|
|
|
‡a
novelαcalcitoningenerelatedpeptideanalogueprotectsagainstendorgandamageinexperimentalhypertensioncardiachypertrophyandheartfailure
‡A
A Novel α-Calcitonin Gene-Related Peptide Analogue Protects Against End-Organ Damage in Experimental Hypertension, Cardiac Hypertrophy, and Heart Failure.
‡9
1
|
919
|
|
|
‡a
chemicalgeneticapproachrevealsthatp38alphamapkactivationbydiphosphorylationaggravatesmyocardialinfarctionandispreventedbythedirectbindingofsb203580
‡A
A chemical genetic approach reveals that p38alpha MAPK activation by diphosphorylation aggravates myocardial infarction and is prevented by the direct binding of SB203580.
‡9
1
|
919
|
|
|
‡a
movinginextremeenvironmentsinertgasnarcosisandunderwateractivities
‡A
Moving in extreme environments: inert gas narcosis and underwater activities.
‡9
1
|
919
|
|
|
‡a
myocardialstressremodellingafterregionalinfarctionisindependentofglycogensynthasekinase3inactivation
‡A
Myocardial stress remodelling after regional infarction is independent of glycogen synthase kinase-3 inactivation.
‡9
1
|
919
|
|
|
‡a
noninvasivewholebodydetectionofcomplementactivationusingradionuclideimaginginamousemodelofmyocardialischaemiareperfusioninjury
‡A
Non-Invasive whole-body detection of complement activation using radionuclide imaging in a mouse model of myocardial ischaemia-reperfusion injury.
‡9
1
|
919
|
|
|
‡a
overexpressionofthetranscriptionfactorhand1causespredispositiontowardsarrhythmiainmice
‡A
Overexpression of the transcription factor Hand1 causes predisposition towards arrhythmia in mice.
‡9
1
|
919
|
|
|
‡a
patentforamenovaleandscubadivingapracticalguideforphysiciansonwhentoreferforscreening
‡A
Patent foramen ovale and scuba diving: a practical guide for physicians on when to refer for screening
‡9
1
|
919
|
|
|
‡a
postinfarctionremodelingisindependentofmitogenactivatedproteinkinasekinase3
‡A
Post-infarction remodeling is independent of mitogen-activated protein kinase kinase 3
‡9
1
|
919
|
|
|
‡a
postinfarctionremodelingisindependentofmitogenactivatedproteinkinasekinase3mkk3
‡A
Post-infarction remodeling is independent of mitogen-activated protein kinase kinase 3 (MKK3)
‡9
1
|
919
|
|
|
‡a
pravastatintherapyduringpreeclampsiapreventslongtermadversehealtheffectsinmice
‡A
Pravastatin therapy during preeclampsia prevents long-term adverse health effects in mice.
‡9
1
|
919
|
|
|
‡a
responsetoroleofhyperleptinemiaintheregulationofbloodpressureandcardiacfunction
‡A
Response to role of hyperleptinemia in the regulation of blood pressure and cardiac function
‡9
1
|
919
|
|
|
‡a
targetingofmannanbindinglectinassociatedserineprotease2confersprotectionfrommyocardialandgastrointestinalischemiareperfusioninjury
‡A
Targeting of mannan-binding lectin-associated serine protease-2 confers protection from myocardial and gastrointestinal ischemia/reperfusion injury.
‡9
1
|
919
|
|
|
‡a
pou4f2brn3btranscriptionfactorisrequiredforthehypertrophicresponsetoangiotensin2intheheart
‡A
The POU4F2/Brn-3b transcription factor is required for the hypertrophic response to angiotensin II in the heart
‡9
1
|
919
|
|
|
‡a
tab1p38αcomplexaggravatesmyocardialinjuryandcanbetargetedbysmallmolecules
‡A
The TAB1-p38α complex aggravates myocardial injury and can be targeted by small molecules
‡9
1
|
919
|
|
|
‡a
thymosinβ4facilitatesepicardialneovascularizationoftheinjuredadultheart
‡A
Thymosin β4 facilitates epicardial neovascularization of the injured adult heart
‡9
1
|
919
|
|
|
‡a
tissueacidosisdoesnotmediatethehypoxiaselectivityofcu
‡A
Tissue acidosis does not mediate the hypoxia selectivity of [Cu][Cu
‡9
1
|
919
|
|
|
‡a
tissueacidosisdoesnotmediatethehypoxiaselectivityofintheisolatedperfusedratheart
‡A
Tissue acidosis does not mediate the hypoxia selectivity of [Cu][Cu(ATSM)] in the isolated perfused rat heart
‡9
1
|
919
|
|
|
‡a
trappedplateletsactivatedinischemiainitiateventricularfibrillation
‡A
Trapped platelets activated in ischemia initiate ventricular fibrillation
‡9
1
|
919
|
|
|
‡a
mechanismandconsequenceoftheautoactivationofp38αmitogenactivatedproteinkinasepromotedbytab1
‡A
Mechanism and consequence of the autoactivation of p38α mitogen-activated protein kinase promoted by TAB1
‡9
1
|
919
|
|
|
‡a
lossofproteinkinasenovel1pkn1isassociatedwithmildsystolicanddiastoliccontractiledysfunctionincreasedphospholambanthr17phosphorylationandexacerbatedischaemiareperfusioninjury
‡A
Loss of Protein Kinase Novel 1 (PKN1) is associated with mild systolic and diastolic contractile dysfunction, increased phospholamban Thr17 phosphorylation, and exacerbated ischaemia-reperfusion injury
‡9
1
|
919
|
|
|
‡a
investigatingthepotentialroleoftrpa1inlocomotionandcardiovascularcontrolduringhypertension
‡A
Investigating the potential role of TRPA1 in locomotion and cardiovascular control during hypertension.
‡9
1
|
946
|
|
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‡a
0 0 lived 0 0
‡9
1
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