VIAF

Virtual International Authority File

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Leader     00000nz a2200037n 45 0
001     WKP|Q89000243  (VIAF cluster)  (Authority/Source Record)
003     WKP
005     20241221010737.0
008     241221nneanz||abbn n and d
035 ‎‡a  (WKP)Q89000243‏
024 ‎‡a  0000-0003-1975-8033‏ ‎‡2  orcid‏
035 ‎‡a  (OCoLC)Q89000243‏
100 0 ‎‡a  Rodrigo Pacheco Silva-Aguiar‏ ‎‡c  investigador mèdic‏ ‎‡9  ca‏
400 0 ‎‡a  Rodrigo Pacheco Silva-Aguiar‏ ‎‡c  researcher‏ ‎‡9  en‏
400 0 ‎‡a  Rodrigo Pacheco Silva-Aguiar‏ ‎‡c  onderzoeker‏ ‎‡9  nl‏
400 0 ‎‡a  Rodrigo Pacheco Silva-Aguiar‏ ‎‡c  investigador médico‏ ‎‡9  es‏
670 ‎‡a  Author's High Glucose Reduces Megalin-Mediated Albumin Endocytosis in Renal Proximal Tubule Cells through Protein Kinase B O-GlcNAcylation.‏
670 ‎‡a  Author's IL-4 Receptor α Chain Protects the Kidney Against Tubule-Interstitial Injury Induced by Albumin Overload‏
670 ‎‡a  Author's Kinins Released by Erythrocytic Stages of Enhance Adhesion of Infected Erythrocytes to Endothelial Cells and Increase Blood Brain Barrier Permeability via Activation of Bradykinin Receptors‏
670 ‎‡a  Author's Lithium ameliorates tubule-interstitial injury through activation of the mTORC2/protein kinase B pathway‏
670 ‎‡a  Author's O-GlcNAcylation reduces proximal tubule protein reabsorption and promotes proteinuria in spontaneously hypertensive rats‏
670 ‎‡a  Author's The angiotensin II/AT1 receptor pathway mediates malaria-induced acute kidney injury‏
670 ‎‡a  Author's The renin-angiotensin-aldosterone system: Role in pathogenesis and potential therapeutic target in COVID-19‏
909 ‎‡a  (orcid) 0000000319758033‏ ‎‡9  1‏
919 ‎‡a  reninangiotensinaldosteronesystemroleinpathogenesisandpotentialtherapeutictargetincovid19‏ ‎‡A  The renin-angiotensin-aldosterone system: Role in pathogenesis and potential therapeutic target in COVID-19‏ ‎‡9  1‏
919 ‎‡a  angiotensin2at1receptorpathwaymediatesmalariainducedacutekidneyinjury‏ ‎‡A  The angiotensin II/AT1 receptor pathway mediates malaria-induced acute kidney injury‏ ‎‡9  1‏
919 ‎‡a  glcnacylationreducesproximaltubuleproteinreabsorptionandpromotesproteinuriainspontaneouslyhypertensiverats‏ ‎‡A  O-GlcNAcylation reduces proximal tubule protein reabsorption and promotes proteinuria in spontaneously hypertensive rats‏ ‎‡9  1‏
919 ‎‡a  lithiumamelioratestubuleinterstitialinjurythroughactivationofthemtorc2proteinkinasebpathway‏ ‎‡A  Lithium ameliorates tubule-interstitial injury through activation of the mTORC2/protein kinase B pathway‏ ‎‡9  1‏
919 ‎‡a  kininsreleasedbyerythrocyticstagesofenhanceadhesionofinfectederythrocytestoendothelialcellsandincreasebloodbrainbarrierpermeabilityviaactivationofbradykininreceptors‏ ‎‡A  Kinins Released by Erythrocytic Stages of Enhance Adhesion of Infected Erythrocytes to Endothelial Cells and Increase Blood Brain Barrier Permeability via Activation of Bradykinin Receptors‏ ‎‡9  1‏
919 ‎‡a  4receptorαchainprotectsthekidneyagainsttubuleinterstitialinjuryinducedbyalbuminoverload‏ ‎‡A  IL-4 Receptor α Chain Protects the Kidney Against Tubule-Interstitial Injury Induced by Albumin Overload‏ ‎‡9  1‏
919 ‎‡a  highglucosereducesmegalinmediatedalbuminendocytosisinrenalproximaltubulecellsthroughproteinkinaseboglcnacylation‏ ‎‡A  High Glucose Reduces Megalin-Mediated Albumin Endocytosis in Renal Proximal Tubule Cells through Protein Kinase B O-GlcNAcylation.‏ ‎‡9  1‏
996 ‎‡2  NII|DA09639250
996 ‎‡2  LC|no2004026894
996 ‎‡2  DBC|87097919400999
996 ‎‡2  LC|nr 96001424
996 ‎‡2  LC|n 2023254517
996 ‎‡2  ISNI|0000000059451632
996 ‎‡2  LC|n 2017251130
996 ‎‡2  ISNI|0000000055067991
996 ‎‡2  ISNI|0000000043585222
996 ‎‡2  LC|n 2005177903
996 ‎‡2  LC|n 2009212896
996 ‎‡2  BNE|XX824414
996 ‎‡2  PTBNP|1779312
996 ‎‡2  BLBNB|001112793
996 ‎‡2  DNB|1121819214
996 ‎‡2  LC|n 2017250684
996 ‎‡2  BLBNB|000228632
996 ‎‡2  ISNI|0000000034871462
996 ‎‡2  BLBNB|001511880
996 ‎‡2  LC|nb2001087725
996 ‎‡2  DNB|1214392083
997 ‎‡a  0 0 lived 0 0‏ ‎‡9  1‏