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Leader
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00000nz a2200037n 45 0 |
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WKP|Q97872828
(VIAF cluster)
(Authority/Source Record)
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WKP |
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20241221010904.0 |
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035
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(WKP)Q97872828
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024
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0000-0002-5499-3082
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orcid
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035
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(OCoLC)Q97872828
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100
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Cristian A. Lasagna Reeves
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professor of anatomy, cell biology & physiology
‡9
en
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375
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1
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iso5218
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400
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Cristian A Lasagna-Reeves
‡c
onderzoeker
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nl
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670
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Author's A native interactor scaffolds and stabilizes toxic ATAXIN-1 oligomers in SCA1.
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670
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Author's A1 reactive astrocytes and a loss of TREM2 are associated with an early stage of pathology in a mouse model of cerebral amyloid angiopathy
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670
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Author's Amyloid-β oligomers as a template for secondary amyloidosis in Alzheimer's disease
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670
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Author's Ataxin-1 oligomers induce local spread of pathology and decreasing them by passive immunization slows Spinocerebellar ataxia type 1 phenotypes.
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670
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Author's Characterization of tau oligomeric seeds in progressive supranuclear palsy
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670
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Author's Dual role of p53 amyloid formation in cancer; loss of function and gain of toxicity.
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670
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Author's Preparation and Characterization of Neurotoxic Tau Oligomers
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670
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Author's The formation of tau pore-like structures is prevalent and cell specific: possible implications for the disease phenotypes
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670
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Author's Vascular amyloid accumulation alters the gabaergic synapse and induces hyperactivity in a model of cerebral amyloid angiopathy
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909
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(orcid) 0000000254993082
‡9
1
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919
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‡a
ataxin1oligomersinducelocalspreadofpathologyanddecreasingthembypassiveimmunizationslowsspinocerebellarataxiatype1phenotypes
‡A
Ataxin-1 oligomers induce local spread of pathology and decreasing them by passive immunization slows Spinocerebellar ataxia type 1 phenotypes.
‡9
1
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919
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‡a
vascularamyloidaccumulationaltersthegabaergicsynapseandinduceshyperactivityinamodelofcerebralamyloidangiopathy
‡A
Vascular amyloid accumulation alters the gabaergic synapse and induces hyperactivity in a model of cerebral amyloid angiopathy
‡9
1
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919
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‡a
formationoftauporelikestructuresisprevalentandcellspecificpossibleimplicationsforthediseasephenotypes
‡A
The formation of tau pore-like structures is prevalent and cell specific: possible implications for the disease phenotypes
‡9
1
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919
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‡a
preparationandcharacterizationofneurotoxictauoligomers
‡A
Preparation and Characterization of Neurotoxic Tau Oligomers
‡9
1
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919
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‡a
dualroleofp53amyloidformationincancerlossoffunctionandgainoftoxicity
‡A
Dual role of p53 amyloid formation in cancer; loss of function and gain of toxicity.
‡9
1
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919
|
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‡a
characterizationoftauoligomericseedsinprogressivesupranuclearpalsy
‡A
Characterization of tau oligomeric seeds in progressive supranuclear palsy
‡9
1
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919
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‡a
amyloidβoligomersasatemplateforsecondaryamyloidosisinalzheimersdisease
‡A
Amyloid-β oligomers as a template for secondary amyloidosis in Alzheimer's disease
‡9
1
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919
|
|
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‡a
a1reactiveastrocytesandalossoftrem2areassociatedwithanearlystageofpathologyinamousemodelofcerebralamyloidangiopathy
‡A
A1 reactive astrocytes and a loss of TREM2 are associated with an early stage of pathology in a mouse model of cerebral amyloid angiopathy
‡9
1
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|
919
|
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‡a
nativeinteractorscaffoldsandstabilizestoxicataxin1oligomersinsca1
‡A
A native interactor scaffolds and stabilizes toxic ATAXIN-1 oligomers in SCA1.
‡9
1
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946
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‡a
b
‡9
1
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996
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ISNI|0000000024222792
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BIBSYS|5022617
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