VIAF

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Leader     00000nz a2200037n 45 0
001     WKP|Q88947288  (VIAF cluster)  (Authority/Source Record)
003     WKP
005     20241121000115.0
008     241121nneanz||abbn n and d
035 ‎‡a  (WKP)Q88947288‏
024 ‎‡a  0000-0002-6895-0024‏ ‎‡2  orcid‏
035 ‎‡a  (OCoLC)Q88947288‏
100 0 ‎‡a  Kai Li‏ ‎‡c  researcher (ORCID 0000-0002-6895-0024)‏ ‎‡9  en‏
670 ‎‡a  Author's Anlotinib suppresses lymphangiogenesis and lymphatic metastasis in lung adenocarcinoma through a process potentially involving VEGFR-3 signaling‏
670 ‎‡a  Author's Bevacizumab promotes active biological behaviors of human umbilical vein endothelial cells by activating TGFβ1 pathways via off-VEGF signaling‏
670 ‎‡a  Author's CD31-labeled circulating endothelial cells as predictor in anlotinib-treated non-small-cell lung cancer: Analysis on ALTER-0303 study.‏
670 ‎‡a  Author's Clinical importance of VEGFC and PD-L1 co-expression in lung adenocarcinoma patients‏
670 ‎‡a  Author's Effect of anlotinib as a third- or further-line therapy in advanced non-small cell lung cancer patients with different histologic types: Subgroup analysis in the ALTER0303 trial‏
670 ‎‡a  Author's Tumor necrosis factor superfamily 15 promotes lymphatic metastasis via upregulation of vascular endothelial growth factor-C in a mouse model of lung cancer‏
909 ‎‡a  (orcid) 0000000268950024‏ ‎‡9  1‏
919 ‎‡a  bevacizumabpromotesactivebiologicalbehaviorsofhumanumbilicalveinendothelialcellsbyactivatingtgfβ1pathwaysviaoffvegfsignaling‏ ‎‡A  Bevacizumab promotes active biological behaviors of human umbilical vein endothelial cells by activating TGFβ1 pathways via off-VEGF signaling‏ ‎‡9  1‏
919 ‎‡a  tumornecrosisfactorsuperfamily15promoteslymphaticmetastasisviaupregulationofvascularendothelialgrowthfactor100inamousemodeloflungcancer‏ ‎‡A  Tumor necrosis factor superfamily 15 promotes lymphatic metastasis via upregulation of vascular endothelial growth factor-C in a mouse model of lung cancer‏ ‎‡9  1‏
919 ‎‡a  clinicalimportanceofvegfcandpdl1coexpressioninlungadenocarcinomapatients‏ ‎‡A  Clinical importance of VEGFC and PD-L1 co-expression in lung adenocarcinoma patients‏ ‎‡9  1‏
919 ‎‡a  anlotinibsuppresseslymphangiogenesisandlymphaticmetastasisinlungadenocarcinomathroughaprocesspotentiallyinvolvingvegfr3signaling‏ ‎‡A  Anlotinib suppresses lymphangiogenesis and lymphatic metastasis in lung adenocarcinoma through a process potentially involving VEGFR-3 signaling‏ ‎‡9  1‏
919 ‎‡a  effectofanlotinibasa3orfurtherlinetherapyinadvancednonsmallcelllungcancerpatientswithdifferenthistologictypessubgroupanalysisinthealter0303trial‏ ‎‡A  Effect of anlotinib as a third- or further-line therapy in advanced non-small cell lung cancer patients with different histologic types: Subgroup analysis in the ALTER0303 trial‏ ‎‡9  1‏
919 ‎‡a  cd31labeledcirculatingendothelialcellsaspredictorinanlotinibtreatednonsmallcelllungcanceranalysisonalter0303study‏ ‎‡A  CD31-labeled circulating endothelial cells as predictor in anlotinib-treated non-small-cell lung cancer: Analysis on ALTER-0303 study.‏ ‎‡9  1‏
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