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001 | WKP|Q88947288 (VIAF cluster) (Authority/Source Record) | ||
003 | WKP | ||
005 | 20241121000115.0 | ||
008 | 241121nneanz||abbn n and d | ||
035 | ‡a (WKP)Q88947288 | ||
024 | ‡a 0000-0002-6895-0024 ‡2 orcid | ||
035 | ‡a (OCoLC)Q88947288 | ||
100 | 0 | ‡a Kai Li ‡c researcher (ORCID 0000-0002-6895-0024) ‡9 en | |
670 | ‡a Author's Anlotinib suppresses lymphangiogenesis and lymphatic metastasis in lung adenocarcinoma through a process potentially involving VEGFR-3 signaling | ||
670 | ‡a Author's Bevacizumab promotes active biological behaviors of human umbilical vein endothelial cells by activating TGFβ1 pathways via off-VEGF signaling | ||
670 | ‡a Author's CD31-labeled circulating endothelial cells as predictor in anlotinib-treated non-small-cell lung cancer: Analysis on ALTER-0303 study. | ||
670 | ‡a Author's Clinical importance of VEGFC and PD-L1 co-expression in lung adenocarcinoma patients | ||
670 | ‡a Author's Effect of anlotinib as a third- or further-line therapy in advanced non-small cell lung cancer patients with different histologic types: Subgroup analysis in the ALTER0303 trial | ||
670 | ‡a Author's Tumor necrosis factor superfamily 15 promotes lymphatic metastasis via upregulation of vascular endothelial growth factor-C in a mouse model of lung cancer | ||
909 | ‡a (orcid) 0000000268950024 ‡9 1 | ||
919 | ‡a bevacizumabpromotesactivebiologicalbehaviorsofhumanumbilicalveinendothelialcellsbyactivatingtgfβ1pathwaysviaoffvegfsignaling ‡A Bevacizumab promotes active biological behaviors of human umbilical vein endothelial cells by activating TGFβ1 pathways via off-VEGF signaling ‡9 1 | ||
919 | ‡a tumornecrosisfactorsuperfamily15promoteslymphaticmetastasisviaupregulationofvascularendothelialgrowthfactor100inamousemodeloflungcancer ‡A Tumor necrosis factor superfamily 15 promotes lymphatic metastasis via upregulation of vascular endothelial growth factor-C in a mouse model of lung cancer ‡9 1 | ||
919 | ‡a clinicalimportanceofvegfcandpdl1coexpressioninlungadenocarcinomapatients ‡A Clinical importance of VEGFC and PD-L1 co-expression in lung adenocarcinoma patients ‡9 1 | ||
919 | ‡a anlotinibsuppresseslymphangiogenesisandlymphaticmetastasisinlungadenocarcinomathroughaprocesspotentiallyinvolvingvegfr3signaling ‡A Anlotinib suppresses lymphangiogenesis and lymphatic metastasis in lung adenocarcinoma through a process potentially involving VEGFR-3 signaling ‡9 1 | ||
919 | ‡a effectofanlotinibasa3orfurtherlinetherapyinadvancednonsmallcelllungcancerpatientswithdifferenthistologictypessubgroupanalysisinthealter0303trial ‡A Effect of anlotinib as a third- or further-line therapy in advanced non-small cell lung cancer patients with different histologic types: Subgroup analysis in the ALTER0303 trial ‡9 1 | ||
919 | ‡a cd31labeledcirculatingendothelialcellsaspredictorinanlotinibtreatednonsmallcelllungcanceranalysisonalter0303study ‡A CD31-labeled circulating endothelial cells as predictor in anlotinib-treated non-small-cell lung cancer: Analysis on ALTER-0303 study. ‡9 1 | ||
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