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001 | WKP|Q54187461 (VIAF cluster) (Authority/Source Record) | ||
003 | WKP | ||
005 | 20241221010840.0 | ||
008 | 241221nneanz||abbn n and d | ||
035 | ‡a (WKP)Q54187461 | ||
024 | ‡a 0000-0002-9619-7666 ‡2 orcid | ||
024 | ‡a 6602756036 ‡2 scopus | ||
035 | ‡a (OCoLC)Q54187461 | ||
100 | 0 | ‡a Theresa C. Thomas ‡9 ast ‡9 es ‡9 sl | |
375 | ‡a 2 ‡2 iso5218 | ||
400 | 0 | ‡a Theresa C. Thomas ‡c researcher ‡9 en | |
400 | 0 | ‡a Theresa C. Thomas ‡c wetenschapper ‡9 nl | |
670 | ‡a Author's Clinical relevance of midline fluid percussion brain injury: Acute deficits, chronic morbidities and the utility of biomarkers. | ||
670 | ‡a Author's Differential effects of amphetamine isomers on dopamine release in the rat striatum and nucleus accumbens core | ||
670 | ‡a Author's Diffuse brain injury elevates tonic glutamate levels and potassium-evoked glutamate release in discrete brain regions at two days post-injury: an enzyme-based microelectrode array study. | ||
670 | ‡a Author's Diffuse traumatic brain injury affects chronic corticosterone function in the rat | ||
670 | ‡a Author's Disruptions in the regulation of extracellular glutamate by neurons and glia in the rat striatum two days after diffuse brain injury. | ||
670 | ‡a Author's Does time heal all wounds? Experimental diffuse traumatic brain injury results in persisting histopathology in the thalamus. | ||
670 | ‡a Author's Dopamine D4 receptor knockout mice exhibit neurochemical changes consistent with decreased dopamine release | ||
670 | ‡a Author's Elucidating opportunities and pitfalls in the treatment of experimental traumatic brain injury to optimize and facilitate clinical translation. | ||
670 | ‡a Author's Hypersensitive glutamate signaling correlates with the development of late-onset behavioral morbidity in diffuse brain-injured circuitry. | ||
670 | ‡a Author's Platelet-mediated changes to neuronal glutamate receptor expression at sites of microthrombosis following experimental subarachnoid hemorrhage | ||
670 | ‡a Author's The time course of activity-regulated cytoskeletal (ARC) gene and protein expression in the whisker-barrel circuit using two paradigms of whisker stimulation. | ||
670 | ‡a Author's Translational Considerations for Behavioral Impairment and Rehabilitation Strategies after Diffuse Traumatic Brain Injury | ||
670 | ‡a Author's Traumatic brain injury alters long-term hippocampal neuron morphology in juvenile, but not immature, rats. | ||
909 | ‡a (scopus) 6602756036 ‡9 1 | ||
909 | ‡a (orcid) 0000000296197666 ‡9 1 | ||
919 | ‡a traumaticbraininjuryalterslongtermhippocampalneuronmorphologyinjuvenilebutnotimmaturerats ‡A Traumatic brain injury alters long-term hippocampal neuron morphology in juvenile, but not immature, rats. ‡9 1 | ||
919 | ‡a translationalconsiderationsforbehavioralimpairmentandrehabilitationstrategiesafterdiffusetraumaticbraininjury ‡A Translational Considerations for Behavioral Impairment and Rehabilitation Strategies after Diffuse Traumatic Brain Injury ‡9 1 | ||
919 | ‡a timecourseofactivityregulatedcytoskeletalarcgeneandproteinexpressioninthewhiskerbarrelcircuitusing2paradigmsofwhiskerstimulation ‡A The time course of activity-regulated cytoskeletal (ARC) gene and protein expression in the whisker-barrel circuit using two paradigms of whisker stimulation. ‡9 1 | ||
919 | ‡a plateletmediatedchangestoneuronalglutamatereceptorexpressionatsitesofmicrothrombosisfollowingexperimentalsubarachnoidhemorrhage ‡A Platelet-mediated changes to neuronal glutamate receptor expression at sites of microthrombosis following experimental subarachnoid hemorrhage ‡9 1 | ||
919 | ‡a hypersensitiveglutamatesignalingcorrelateswiththedevelopmentoflateonsetbehavioralmorbidityindiffusebraininjuredcircuitry ‡A Hypersensitive glutamate signaling correlates with the development of late-onset behavioral morbidity in diffuse brain-injured circuitry. ‡9 1 | ||
919 | ‡a elucidatingopportunitiesandpitfallsinthetreatmentofexperimentaltraumaticbraininjurytooptimizeandfacilitateclinicaltranslation ‡A Elucidating opportunities and pitfalls in the treatment of experimental traumatic brain injury to optimize and facilitate clinical translation. ‡9 1 | ||
919 | ‡a dopamined4receptorknockoutmiceexhibitneurochemicalchangesconsistentwithdecreaseddopaminerelease ‡A Dopamine D4 receptor knockout mice exhibit neurochemical changes consistent with decreased dopamine release ‡9 1 | ||
919 | ‡a doestimehealallwoundsexperimentaldiffusetraumaticbraininjuryresultsinpersistinghistopathologyinthethalamus ‡A Does time heal all wounds? Experimental diffuse traumatic brain injury results in persisting histopathology in the thalamus. ‡9 1 | ||
919 | ‡a disruptionsintheregulationofextracellularglutamatebyneuronsandgliaintheratstriatum2daysafterdiffusebraininjury ‡A Disruptions in the regulation of extracellular glutamate by neurons and glia in the rat striatum two days after diffuse brain injury. ‡9 1 | ||
919 | ‡a diffusetraumaticbraininjuryaffectschroniccorticosteronefunctionintherat ‡A Diffuse traumatic brain injury affects chronic corticosterone function in the rat ‡9 1 | ||
919 | ‡a diffusebraininjuryelevatestonicglutamatelevelsandpotassiumevokedglutamatereleaseindiscretebrainregionsat2dayspostinjuryanenzymebasedmicroelectrodearraystudy ‡A Diffuse brain injury elevates tonic glutamate levels and potassium-evoked glutamate release in discrete brain regions at two days post-injury: an enzyme-based microelectrode array study. ‡9 1 | ||
919 | ‡a differentialeffectsofamphetamineisomersondopaminereleaseintheratstriatumandnucleusaccumbenscore ‡A Differential effects of amphetamine isomers on dopamine release in the rat striatum and nucleus accumbens core ‡9 1 | ||
919 | ‡a clinicalrelevanceofmidlinefluidpercussionbraininjuryacutedeficitschronicmorbiditiesandtheutilityofbiomarkers ‡A Clinical relevance of midline fluid percussion brain injury: Acute deficits, chronic morbidities and the utility of biomarkers. ‡9 1 | ||
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