VIAF

Virtual International Authority File

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Leader 00000nz a2200037n 45 0
001 WKP|Q54867176 (VIAF cluster) (Authority/Source Record)
003 WKP
005 20241120235922.0
008 241120nneanz||abbn n and d
035 ‎‡a (WKP)Q54867176‏
024 ‎‡a 0000-0002-5181-2135‏ ‎‡2 orcid‏
024 ‎‡a 7407089703‏ ‎‡2 scopus‏
035 ‎‡a (OCoLC)Q54867176‏
100 0 ‎‡a Wan Lee‏ ‎‡9 ast‏ ‎‡9 es‏ ‎‡9 sl‏
400 0 ‎‡a Wan Lee‏ ‎‡c researcher (ORCID 0000-0002-5181-2135)‏ ‎‡9 en‏
400 0 ‎‡a Wan Lee‏ ‎‡c wetenschapper‏ ‎‡9 nl‏
670 ‎‡a Author's A Synthetic Peptide Corresponding to the GLUT4 C-terminal Cytoplasmic Domain Causes Insulin-like Glucose Transport Stimulation and GLUT4 Recruitment in Rat Adipocytes‏
670 ‎‡a Author's Association of carboxyl esterase with facilitative glucose transporter isoform 4‏
670 ‎‡a Author's Association of carboxyl esterase with facilitative glucose transporter isoform 4 (GLUT4) intracellular compartments in rat adipocytes and its possible role in insulin-induced GLUT4 recruitment.‏
670 ‎‡a Author's C1q tumor necrosis factor Alpha -related protein isoform 5 is increased in mitochondrial DNA-depleted myocytes and activates AMP-activated protein kinase‏
670 ‎‡a Author's C1q tumor necrosis factor α-related protein isoform 5 attenuates palmitate-induced DNA fragmentation in myocytes through an AMPK-dependent mechanism‏
670 ‎‡a Author's C1qTNF-related protein-6 mediates fatty acid oxidation via the activation of the AMP-activated protein kinase‏
670 ‎‡a Author's Cadmium induces impaired glucose tolerance in rat by down-regulating GLUT4 expression in adipocytes.‏
670 ‎‡a Author's Combination gene therapy using multidrug resistance‏
670 ‎‡a Author's Combination gene therapy using multidrug resistance (MDR1) gene shRNA and herpes simplex virus-thymidine kinase‏
670 ‎‡a Author's CTRP5 ameliorates palmitate-induced apoptosis and insulin resistance through activation of AMPK and fatty acid oxidation.‏
670 ‎‡a Author's Dangnyohwan improves glucose utilization and reduces insulin resistance by increasing the adipocyte-specific GLUT4 expression in Otsuka Long-Evans Tokushima Fatty rats.‏
670 ‎‡a Author's Data for differentially expressed microRNAs in saturated fatty acid palmitate-treated HepG2 cells‏
670 ‎‡a Author's Data on the decreased expression of FOXO1 by miR-1271 in HepG2 hepatocytes‏
670 ‎‡a Author's Data on the effect of miR-15b on the expression of INSR in murine C2C12 myocytes‏
670 ‎‡a Author's Data on the expression and insulin-stimulated phosphorylation of IRS-1 by miR-96 in L6-GLUT4myc myocytes‏
670 ‎‡a Author's Data on the expression of PEPCK in HepG2 hepatocytes transfected with miR-195.‏
670 ‎‡a Author's Dataset on the identification of differentially expressed genes by annealing control primer-based PCR in mitochondrial DNA-depleted myocytes.‏
670 ‎‡a Author's Depletion of mitochondrial DNA causes impaired glucose utilization and insulin resistance in L6 GLUT4myc myocytes.‏
670 ‎‡a Author's Depletion of mitochondrial DNA up-regulates the expression of MDR1 gene via an increase in mRNA stability.‏
670 ‎‡a Author's EHD2 interacts with the insulin-responsive glucose transporter (GLUT4) in rat adipocytes and may participate in insulin-induced GLUT4 recruitment.‏
670 ‎‡a Author's Ets-2 is involved in transcriptional regulation of C1qTNF-related protein 5 in muscle cells‏
670 ‎‡a Author's Exosome-derived microRNAs in cancer metabolism: possible implications in cancer diagnostics and therapy.‏
670 ‎‡a Author's Genetic risk for metabolic syndrome: examination of candidate gene polymorphisms related to lipid metabolism in Japanese people.‏
670 ‎‡a Author's Glucose transporters and insulin action: some insights into diabetes management.‏
670 ‎‡a Author's Implications of microRNAs in the pathogenesis of diabetes.‏
670 ‎‡a Author's Induction of miR-29a by saturated fatty acids impairs insulin signaling and glucose uptake through translational repression of IRS-1 in myocytes.‏
670 ‎‡a Author's Induction of miR-96 by Dietary Saturated Fatty Acids Exacerbates Hepatic Insulin Resistance through the Suppression of INSR and IRS-1.‏
670 ‎‡a Author's MicroRNA-126 suppresses mesothelioma malignancy by targeting IRS1 and interfering with the mitochondrial function‏
670 ‎‡a Author's MicroRNA expression analysis in the liver of high fat diet-induced obese mice‏
670 ‎‡a Author's MiR-1271 upregulated by saturated fatty acid palmitate provokes impaired insulin signaling by repressing INSR and IRS-1 expression in HepG2 cells‏
670 ‎‡a Author's MiR-96-5p Induced by Palmitic Acid Suppresses the Myogenic Differentiation of C2C12 Myoblasts by Targeting FHL1‏
670 ‎‡a Author's Modulation of GLUT4 and GLUT1 recycling by insulin in rat adipocytes: kinetic analysis based on the involvement of multiple intracellular compartments.‏
670 ‎‡a Author's N-Acetylated Alpha -linked acidic dipeptidase expressed in rat adipocytes is localized in the insulin-responsive glucose transporter (GLUT4) intracellular compartments and involved in the insulin-stimulated GLUT4 recruitment.‏
670 ‎‡a Author's Obesity-induced miR-15b is linked causally to the development of insulin resistance through the repression of the insulin receptor in hepatocytes.‏
670 ‎‡a Author's Proteomic analysis of cellular change involved in mitochondria-to-nucleus communication in L6 GLUT4myc myocytes‏
670 ‎‡a Author's Regulation of the transcriptional activation of CTRP3 in chondrocytes by c-Jun.‏
670 ‎‡a Author's Saturated fatty acid-induced miR-195 impairs insulin signaling and glycogen metabolism in HepG2 cells.‏
670 ‎‡a Author's Saturated fatty acids-induced miR-424-5p aggravates insulin resistance via targeting insulin receptor in hepatocytes‏
670 ‎‡a Author's Sfrp2 is a transcriptional target of SREBP-1 in mouse chondrogenic cells.‏
670 ‎‡a Author's The hepatocyte glucose-6-phosphatase subcomponent T3: its relationship to GLUT2.‏
670 ‎‡a Author's The induction of microRNA targeting IRS-1 is involved in the development of insulin resistance under conditions of mitochondrial dysfunction in hepatocytes‏
670 ‎‡a Author's The induction of miR-96 by mitochondrial dysfunction causes impaired glycogen synthesis through translational repression of IRS-1 in SK-Hep1 cells.‏
909 ‎‡a (orcid) 0000000251812135‏ ‎‡9 1‏
909 ‎‡a (scopus) 7407089703‏ ‎‡9 1‏
919 ‎‡a syntheticpeptidecorrespondingtotheglut4100terminalcytoplasmicdomaincausesinsulinlikeglucosetransportstimulationandglut4recruitmentinratadipocytes‏ ‎‡A A Synthetic Peptide Corresponding to the GLUT4 C-terminal Cytoplasmic Domain Causes Insulin-like Glucose Transport Stimulation and GLUT4 Recruitment in Rat Adipocytes‏ ‎‡9 1‏
919 ‎‡a associationofcarboxylesterasewithfacilitativeglucosetransporterisoform4‏ ‎‡A Association of carboxyl esterase with facilitative glucose transporter isoform 4‏ ‎‡9 1‏
919 ‎‡a associationofcarboxylesterasewithfacilitativeglucosetransporterisoform4glut4intracellularcompartmentsinratadipocytesanditspossibleroleininsulininducedglut4recruitment‏ ‎‡A Association of carboxyl esterase with facilitative glucose transporter isoform 4 (GLUT4) intracellular compartments in rat adipocytes and its possible role in insulin-induced GLUT4 recruitment.‏ ‎‡9 1‏
919 ‎‡a c1qtumornecrosisfactor Alpha relatedproteinisoform5isincreasedinmitochondrialdnadepletedmyocytesandactivatesampactivatedproteinkinase‏ ‎‡A C1q tumor necrosis factor Alpha -related protein isoform 5 is increased in mitochondrial DNA-depleted myocytes and activates AMP-activated protein kinase‏ ‎‡9 1‏
919 ‎‡a c1qtumornecrosisfactorαrelatedproteinisoform5attenuatespalmitateinduceddnafragmentationinmyocytesthroughanampkdependentmechanism‏ ‎‡A C1q tumor necrosis factor α-related protein isoform 5 attenuates palmitate-induced DNA fragmentation in myocytes through an AMPK-dependent mechanism‏ ‎‡9 1‏
919 ‎‡a c1qtnfrelatedprotein6mediatesfattyacidoxidationviatheactivationoftheampactivatedproteinkinase‏ ‎‡A C1qTNF-related protein-6 mediates fatty acid oxidation via the activation of the AMP-activated protein kinase‏ ‎‡9 1‏
919 ‎‡a cadmiuminducesimpairedglucosetoleranceinratbydownregulatingglut4expressioninadipocytes‏ ‎‡A Cadmium induces impaired glucose tolerance in rat by down-regulating GLUT4 expression in adipocytes.‏ ‎‡9 1‏
919 ‎‡a combinationgenetherapyusingmultidrugresistance‏ ‎‡A Combination gene therapy using multidrug resistance‏ ‎‡9 1‏
919 ‎‡a combinationgenetherapyusingmultidrugresistancemdr1geneshrnaandherpessimplexvirusthymidinekinase‏ ‎‡A Combination gene therapy using multidrug resistance (MDR1) gene shRNA and herpes simplex virus-thymidine kinase‏ ‎‡9 1‏
919 ‎‡a ctrp5amelioratespalmitateinducedapoptosisandinsulinresistancethroughactivationofampkandfattyacidoxidation‏ ‎‡A CTRP5 ameliorates palmitate-induced apoptosis and insulin resistance through activation of AMPK and fatty acid oxidation.‏ ‎‡9 1‏
919 ‎‡a dangnyohwanimprovesglucoseutilizationandreducesinsulinresistancebyincreasingtheadipocytespecificglut4expressioninotsukalongevanstokushimafattyrats‏ ‎‡A Dangnyohwan improves glucose utilization and reduces insulin resistance by increasing the adipocyte-specific GLUT4 expression in Otsuka Long-Evans Tokushima Fatty rats.‏ ‎‡9 1‏
919 ‎‡a dataonthedecreasedexpressionoffoxo1bymir1271inhepg2hepatocytes‏ ‎‡A Data on the decreased expression of FOXO1 by miR-1271 in HepG2 hepatocytes‏ ‎‡9 1‏
919 ‎‡a dataontheeffectofmir15bontheexpressionofinsrinmurinec2c12myocytes‏ ‎‡A Data on the effect of miR-15b on the expression of INSR in murine C2C12 myocytes‏ ‎‡9 1‏
919 ‎‡a dataontheexpressionandinsulinstimulatedphosphorylationofirs1bymir96inl6glut4mycmyocytes‏ ‎‡A Data on the expression and insulin-stimulated phosphorylation of IRS-1 by miR-96 in L6-GLUT4myc myocytes‏ ‎‡9 1‏
919 ‎‡a dataontheexpressionofpepckinhepg2hepatocytestransfectedwithmir195‏ ‎‡A Data on the expression of PEPCK in HepG2 hepatocytes transfected with miR-195.‏ ‎‡9 1‏
919 ‎‡a datasetontheidentificationofdifferentiallyexpressedgenesbyannealingcontrol1basedpcrinmitochondrialdnadepletedmyocytes‏ ‎‡A Dataset on the identification of differentially expressed genes by annealing control primer-based PCR in mitochondrial DNA-depleted myocytes.‏ ‎‡9 1‏
919 ‎‡a depletionofmitochondrialdnacausesimpairedglucoseutilizationandinsulinresistanceinl6glut4mycmyocytes‏ ‎‡A Depletion of mitochondrial DNA causes impaired glucose utilization and insulin resistance in L6 GLUT4myc myocytes.‏ ‎‡9 1‏
919 ‎‡a depletionofmitochondrialdnaupregulatestheexpressionofmdr1geneviaanincreaseinmrnastability‏ ‎‡A Depletion of mitochondrial DNA up-regulates the expression of MDR1 gene via an increase in mRNA stability.‏ ‎‡9 1‏
919 ‎‡a ehd2interactswiththeinsulinresponsiveglucosetransporterglut4inratadipocytesandmayparticipateininsulininducedglut4recruitment‏ ‎‡A EHD2 interacts with the insulin-responsive glucose transporter (GLUT4) in rat adipocytes and may participate in insulin-induced GLUT4 recruitment.‏ ‎‡9 1‏
919 ‎‡a ets2isinvolvedintranscriptionalregulationofc1qtnfrelatedprotein5inmusclecells‏ ‎‡A Ets-2 is involved in transcriptional regulation of C1qTNF-related protein 5 in muscle cells‏ ‎‡9 1‏
919 ‎‡a exosomederivedmicrornasincancermetabolismpossibleimplicationsincancerdiagnosticsandtherapy‏ ‎‡A Exosome-derived microRNAs in cancer metabolism: possible implications in cancer diagnostics and therapy.‏ ‎‡9 1‏
919 ‎‡a geneticriskformetabolicsyndromeexaminationofcandidategenepolymorphismsrelatedtolipidmetabolisminjapanesepeople‏ ‎‡A Genetic risk for metabolic syndrome: examination of candidate gene polymorphisms related to lipid metabolism in Japanese people.‏ ‎‡9 1‏
919 ‎‡a glucosetransportersandinsulinactionsomeinsightsintodiabetesmanagement‏ ‎‡A Glucose transporters and insulin action: some insights into diabetes management.‏ ‎‡9 1‏
919 ‎‡a implicationsofmicrornasinthepathogenesisofdiabetes‏ ‎‡A Implications of microRNAs in the pathogenesis of diabetes.‏ ‎‡9 1‏
919 ‎‡a inductionofmir29abysaturatedfattyacidsimpairsinsulinsignalingandglucoseuptakethroughtranslationalrepressionofirs1inmyocytes‏ ‎‡A Induction of miR-29a by saturated fatty acids impairs insulin signaling and glucose uptake through translational repression of IRS-1 in myocytes.‏ ‎‡9 1‏
919 ‎‡a inductionofmir96bydietarysaturatedfattyacidsexacerbateshepaticinsulinresistancethroughthesuppressionofinsrandirs1‏ ‎‡A Induction of miR-96 by Dietary Saturated Fatty Acids Exacerbates Hepatic Insulin Resistance through the Suppression of INSR and IRS-1.‏ ‎‡9 1‏
919 ‎‡a microrna126suppressesmesotheliomamalignancybytargetingirs1andinterferingwiththemitochondrialfunction‏ ‎‡A MicroRNA-126 suppresses mesothelioma malignancy by targeting IRS1 and interfering with the mitochondrial function‏ ‎‡9 1‏
919 ‎‡a micrornaexpressionanalysisintheliverofhighfatdietinducedobesemice‏ ‎‡A MicroRNA expression analysis in the liver of high fat diet-induced obese mice‏ ‎‡9 1‏
919 ‎‡a mir1271upregulatedbysaturatedfattyacidpalmitateprovokesimpairedinsulinsignalingbyrepressinginsrandirs1expressioninhepg2cells‏ ‎‡A MiR-1271 upregulated by saturated fatty acid palmitate provokes impaired insulin signaling by repressing INSR and IRS-1 expression in HepG2 cells‏ ‎‡9 1‏
919 ‎‡a mir965pinducedbypalmiticacidsuppressesthemyogenicdifferentiationofc2c12myoblastsbytargetingfhl1‏ ‎‡A MiR-96-5p Induced by Palmitic Acid Suppresses the Myogenic Differentiation of C2C12 Myoblasts by Targeting FHL1‏ ‎‡9 1‏
919 ‎‡a modulationofglut4andglut1recyclingbyinsulininratadipocyteskineticanalysisbasedontheinvolvementofmultipleintracellularcompartments‏ ‎‡A Modulation of GLUT4 and GLUT1 recycling by insulin in rat adipocytes: kinetic analysis based on the involvement of multiple intracellular compartments.‏ ‎‡9 1‏
919 ‎‡a nacetylated Alpha linkedacidicdipeptidaseexpressedinratadipocytesislocalizedintheinsulinresponsiveglucosetransporterglut4intracellularcompartmentsandinvolvedintheinsulinstimulatedglut4recruitment‏ ‎‡A N-Acetylated Alpha -linked acidic dipeptidase expressed in rat adipocytes is localized in the insulin-responsive glucose transporter (GLUT4) intracellular compartments and involved in the insulin-stimulated GLUT4 recruitment.‏ ‎‡9 1‏
919 ‎‡a obesityinducedmir15bislinkedcausallytothedevelopmentofinsulinresistancethroughtherepressionoftheinsulinreceptorinhepatocytes‏ ‎‡A Obesity-induced miR-15b is linked causally to the development of insulin resistance through the repression of the insulin receptor in hepatocytes.‏ ‎‡9 1‏
919 ‎‡a proteomicanalysisofcellularchangeinvolvedinmitochondriatonucleuscommunicationinl6glut4mycmyocytes‏ ‎‡A Proteomic analysis of cellular change involved in mitochondria-to-nucleus communication in L6 GLUT4myc myocytes‏ ‎‡9 1‏
919 ‎‡a regulationofthetranscriptionalactivationofctrp3inchondrocytesby100jun‏ ‎‡A Regulation of the transcriptional activation of CTRP3 in chondrocytes by c-Jun.‏ ‎‡9 1‏
919 ‎‡a saturatedfattyacidinducedmir195impairsinsulinsignalingandglycogenmetabolisminhepg2cells‏ ‎‡A Saturated fatty acid-induced miR-195 impairs insulin signaling and glycogen metabolism in HepG2 cells.‏ ‎‡9 1‏
919 ‎‡a saturatedfattyacidsinducedmir4245paggravatesinsulinresistanceviatargetinginsulinreceptorinhepatocytes‏ ‎‡A Saturated fatty acids-induced miR-424-5p aggravates insulin resistance via targeting insulin receptor in hepatocytes‏ ‎‡9 1‏
919 ‎‡a sfrp2isatranscriptionaltargetofsrebp1inmousechondrogeniccells‏ ‎‡A Sfrp2 is a transcriptional target of SREBP-1 in mouse chondrogenic cells.‏ ‎‡9 1‏
919 ‎‡a hepatocyteglucose6phosphatasesubcomponentt3itsrelationshiptoglut2‏ ‎‡A The hepatocyte glucose-6-phosphatase subcomponent T3: its relationship to GLUT2.‏ ‎‡9 1‏
919 ‎‡a inductionofmicrornatargetingirs1isinvolvedinthedevelopmentofinsulinresistanceunderconditionsofmitochondrialdysfunctioninhepatocytes‏ ‎‡A The induction of microRNA targeting IRS-1 is involved in the development of insulin resistance under conditions of mitochondrial dysfunction in hepatocytes‏ ‎‡9 1‏
919 ‎‡a inductionofmir96bymitochondrialdysfunctioncausesimpairedglycogensynthesisthroughtranslationalrepressionofirs1inskhep1cells‏ ‎‡A The induction of miR-96 by mitochondrial dysfunction causes impaired glycogen synthesis through translational repression of IRS-1 in SK-Hep1 cells.‏ ‎‡9 1‏
919 ‎‡a datafordifferentiallyexpressedmicrornasinsaturatedfattyacidpalmitatetreatedhepg2cells‏ ‎‡A Data for differentially expressed microRNAs in saturated fatty acid palmitate-treated HepG2 cells‏ ‎‡9 1‏
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998 ‎‡a Wan, Lee‏ ‎‡2 RERO|A028751219‏ ‎‡3 exact name‏