VIAF

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Leader 00000nz a2200037n 45 0
001 WKP|Q54918334 (VIAF cluster) (Authority/Source Record)
003 WKP
005 20241121000325.0
008 241121nneanz||abbn n and d
035 ‎‡a (WKP)Q54918334‏
024 ‎‡a 0000-0002-0079-0773‏ ‎‡2 orcid‏
035 ‎‡a (OCoLC)Q54918334‏
100 0 ‎‡a Jing Wang‏ ‎‡9 ast‏ ‎‡9 es‏ ‎‡9 sl‏
400 0 ‎‡a Jing Wang‏ ‎‡c researcher ORCID: 0000-0002-0079-0773‏ ‎‡9 en‏
400 0 ‎‡a Jing Wang‏ ‎‡c onderzoeker‏ ‎‡9 nl‏
670 ‎‡a Author's A combination of nonoperative treatment modalities used for treatment of keloids‏
670 ‎‡a Author's Pathogenic Streptococcus strains employ novel escape strategy to inhibit bacteriostatic effect mediated by mammalian peptidoglycan recognition protein.‏
670 ‎‡a Author's The ribosomal protein L32-2 (RPL32-2) of S. pombe exhibits a novel extraribosomal function by acting as a potential transcriptional regulator‏
909 ‎‡a (orcid) 0000000200790773‏ ‎‡9 1‏
919 ‎‡a combinationofnonoperativetreatmentmodalitiesusedfortreatmentofkeloids‏ ‎‡A A combination of nonoperative treatment modalities used for treatment of keloids‏ ‎‡9 1‏
919 ‎‡a pathogenicstreptococcusstrainsemploynovelescapestrategytoinhibitbacteriostaticeffectmediatedbymammalianpeptidoglycanrecognitionprotein‏ ‎‡A Pathogenic Streptococcus strains employ novel escape strategy to inhibit bacteriostatic effect mediated by mammalian peptidoglycan recognition protein.‏ ‎‡9 1‏
919 ‎‡a ribosomalproteinl322rpl322ofspombeexhibitsanovelextraribosomalfunctionbyactingasapotentialtranscriptionalregulator‏ ‎‡A The ribosomal protein L32-2 (RPL32-2) of S. pombe exhibits a novel extraribosomal function by acting as a potential transcriptional regulator‏ ‎‡9 1‏
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997 ‎‡a 0 0 lived 0 0‏ ‎‡9 1‏
998 ‎‡a Jing, Wang‏ ‎‡2 NKC|xx0241517‏ ‎‡3 exact name‏
998 ‎‡a Jing, Wang‏ ‎‡2 NTA|410365777‏ ‎‡3 exact name‏
998 ‎‡a Jing, Wang‏ ‎‡2 SUDOC|244956170‏ ‎‡3 exact name‏