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Author's Osteoclasts Derive Predominantly from Bone Marrow-Resident CX3CR1+ Precursor Cells in Homeostasis, whereas Circulating CX3CR1+ Cells Contribute to Osteoclast Development during Fracture Repair
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Author's Protease-Activated Receptor 1 Deletion Causes Enhanced Osteoclastogenesis in Response to Inflammatory Signals through a Notch2-Dependent Mechanism
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cd55regulatesbonemassinmicebymodulatingranklmediatedracsignalingandosteoclastfunction
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CD55 Regulates Bone Mass in Mice by Modulating RANKL-Mediated Rac Signaling and Osteoclast Function
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1
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osteoclastsderivepredominantlyfrombonemarrowresidentcx3cr1+precursorcellsinhomeostasiswhereascirculatingcx3cr1+cellscontributetoosteoclastdevelopmentduringfracturerepair
‡A
Osteoclasts Derive Predominantly from Bone Marrow-Resident CX3CR1+ Precursor Cells in Homeostasis, whereas Circulating CX3CR1+ Cells Contribute to Osteoclast Development during Fracture Repair
‡9
1