VIAF

Virtual International Authority File

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Leader 00000nz a2200037n 45 0
001 WKP|Q90092118 (VIAF cluster) (Authority/Source Record)
003 WKP
005 20241120235913.0
008 241120nneanz||abbn n and d
035 ‎‡a (WKP)Q90092118‏
024 ‎‡a 0000-0002-5284-2213‏ ‎‡2 orcid‏
035 ‎‡a (OCoLC)Q90092118‏
100 0 ‎‡a Jin-Ying Zhang‏ ‎‡c researcher‏ ‎‡9 en‏
400 0 ‎‡a Jin-Ying Zhang‏ ‎‡c wetenschapper‏ ‎‡9 nl‏
670 ‎‡a Author's 12(S)-hydroxyeicosatetraenoic acid impairs vascular endothelial permeability by altering adherens junction phosphorylation levels and affecting the binding and dissociation of its components in high glucose-induced vascular injury‏
670 ‎‡a Author's ALT-to-Lymphocyte Ratio as a Predictor of Long-Term Mortality in Patients with Normal Liver Function Presenting Coronary Artery Disease after Undergoing PCI: A Retrospective Cohort Study‏
670 ‎‡a Author's Amelioration of atherosclerosis in apolipoprotein E-deficient mice by combined RNA interference of lipoprotein-associated phospholipase A2 and YKL-40‏
670 ‎‡a Author's Circulating exosomes repair endothelial cell damage by delivering miR-193a-5p‏
670 ‎‡a Author's COVID-19 and the cardiovascular system‏
670 ‎‡a Author's Evaluation of Mechanical Performances of Stents with 38 mm Length in Long Lesion‏
670 ‎‡a Author's Reply to: 'Interaction between RAAS inhibitors and ACE2 in the context of COVID-19'‏
670 ‎‡a Author's Resting Heart Rate and Long-Term Outcomes in Patients with Percutaneous Coronary Intervention: Results from a 10-Year Follow-Up of the CORFCHD-PCI Study‏
670 ‎‡a Author's Small but significant: Insights and new perspectives of exosomes in cardiovascular disease‏
670 ‎‡a Author's Sodium (±)-5-bromo-2-(α-hydroxypentyl) benzoate ameliorates pressure overload-induced cardiac hypertrophy and dysfunction through inhibiting autophagy‏
909 ‎‡a (orcid) 0000000252842213‏ ‎‡9 1‏
919 ‎‡a replytointeractionbetweenraasinhibitorsandace2inthecontextofcovid19‏ ‎‡A Reply to: 'Interaction between RAAS inhibitors and ACE2 in the context of COVID-19'‏ ‎‡9 1‏
919 ‎‡a covid19andthecardiovascularsystem‏ ‎‡A COVID-19 and the cardiovascular system‏ ‎‡9 1‏
919 ‎‡a circulatingexosomesrepairendothelialcelldamagebydeliveringmir193a5p‏ ‎‡A Circulating exosomes repair endothelial cell damage by delivering miR-193a-5p‏ ‎‡9 1‏
919 ‎‡a ameliorationofatherosclerosisinapolipoproteinedeficientmicebycombinedrnainterferenceoflipoproteinassociatedphospholipasea2andykl40‏ ‎‡A Amelioration of atherosclerosis in apolipoprotein E-deficient mice by combined RNA interference of lipoprotein-associated phospholipase A2 and YKL-40‏ ‎‡9 1‏
919 ‎‡a alttolymphocyteratioasapredictoroflongtermmortalityinpatientswithnormalliverfunctionpresentingcoronaryarterydiseaseafterundergoingpciaretrospectivecohortstudy‏ ‎‡A ALT-to-Lymphocyte Ratio as a Predictor of Long-Term Mortality in Patients with Normal Liver Function Presenting Coronary Artery Disease after Undergoing PCI: A Retrospective Cohort Study‏ ‎‡9 1‏
919 ‎‡a 12shydroxyeicosatetraenoicacidimpairsvascularendothelialpermeabilitybyalteringadherensjunctionphosphorylationlevelsandaffectingthebindinganddissociationofitscomponentsinhighglucoseinducedvascularinjury‏ ‎‡A 12(S)-hydroxyeicosatetraenoic acid impairs vascular endothelial permeability by altering adherens junction phosphorylation levels and affecting the binding and dissociation of its components in high glucose-induced vascular injury‏ ‎‡9 1‏
919 ‎‡a smallbutsignificantinsightsandnewperspectivesofexosomesincardiovasculardisease‏ ‎‡A Small but significant: Insights and new perspectives of exosomes in cardiovascular disease‏ ‎‡9 1‏
919 ‎‡a restingheartrateandlongtermoutcomesinpatientswithpercutaneouscoronaryinterventionresultsfroma10yearfollowupofthecorfchdpcistudy‏ ‎‡A Resting Heart Rate and Long-Term Outcomes in Patients with Percutaneous Coronary Intervention: Results from a 10-Year Follow-Up of the CORFCHD-PCI Study‏ ‎‡9 1‏
919 ‎‡a sodium5bromo2αhydroxypentylbenzoateamelioratespressureoverloadinducedcardiachypertrophyanddysfunctionthroughinhibitingautophagy‏ ‎‡A Sodium (±)-5-bromo-2-(α-hydroxypentyl) benzoate ameliorates pressure overload-induced cardiac hypertrophy and dysfunction through inhibiting autophagy‏ ‎‡9 1‏
919 ‎‡a evaluationofmechanicalperformancesofstentswith382000lengthinlonglesion‏ ‎‡A Evaluation of Mechanical Performances of Stents with 38 mm Length in Long Lesion‏ ‎‡9 1‏
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997 ‎‡a 0 0 lived 0 0‏ ‎‡9 1‏